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Due to PCT’s variance between microbial infections and healthy individuals, it has become a marker to improve identification of bacterial infection and guide antibiotic therapy. [14] The table below is a summary from Schuetz, Albrich, and Mueller, [ 14 ] summarizing the current data of selected, relevant studies investigating PCT in different ...
The main discussion of these abbreviations in the context of drug prescriptions and other medical prescriptions is at List of abbreviations used in medical prescriptions. Some of these abbreviations are best not used, as marked and explained here.
HeV Infection Hendra virus infection HF Heart failure: HFA High-functioning autism: HFMD Hand, foot, and mouth disease: HFRS Hemorrhagic fever with renal syndrome: HI Hearing impaired: HiB disease Haemophilus influenzae type B disease: HIBM Hereditary inclusion body myopathy: HMSN Type III
Pronunciation follows convention outside the medical field, in which acronyms are generally pronounced as if they were a word (JAMA, SIDS), initialisms are generally pronounced as individual letters (DNA, SSRI), and abbreviations generally use the expansion (soln. = "solution", sup. = "superior").
PCT: progesterone challenge test Patient care technician: PCV: packed cell volume (see hematocrit) polycythemia vera: PCWP: pulmonary capillary wedge pressure: PD: Parkinson's disease paroxysmal dyskinesia peritoneal dialysis physical diagnosis personality disorder program director PDA: patent ductus arteriosus posterior descending artery: PDD
Typically in these individuals, a variety of risk factors such as alcohol use disorder and Hepatitis C virus infection co-occur to result in the clinical manifestation of PCT. Type II ("familial") 176100: Patients exhibiting Type II PCT have a specific deficiency in the UROD gene, passed down in an autosomal dominant pattern.
[2] [3] The mechanism of infection is usually spread up the urinary tract. [2] Less often infection occurs through the bloodstream. [1] Diagnosis is typically based on symptoms and supported by urinalysis. [2] If there is no improvement with treatment, medical imaging may be recommended. [2]
Sodium-dependent glucose cotransporters (or sodium-glucose linked transporter, SGLT) are a family of glucose transporter found in the intestinal mucosa (enterocytes) of the small intestine (SGLT1) and the proximal tubule of the nephron (SGLT2 in PCT and SGLT1 in PST). They contribute to renal glucose reabsorption.