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Management of the dawn phenomenon varies by patient and thus should be done with regular assistance from a patient's physician. Some treatment options include, but are not limited to, dietary modifications, increased exercise before breakfast and during the evening, and oral anti-hyperglycemic medications if a patient's HbA1c is > 7%.
Chronic Somogyi rebound is a contested explanation of phenomena of elevated blood sugars experienced by diabetics in the morning. Also called the Somogyi effect and posthypoglycemic hyperglycemia, it is a rebounding high blood sugar that is a response to low blood sugar. [1]
Diabetic ketoacidosis (DKA), most typically seen in those with type 1 diabetes, is triggered by the build-up of chemicals called ketones. These are strongly acidic and a build-up can cause the blood to become acidic. [5] When these levels get too high it essentially poisons the body and causes DKA. [6]
Of course, diabetics should do everything in their power to avoid spikes in these nutrients and maintain healthy levels at all times. That doesn't mean that diabetics can't eat fast food at all ...
Diabetic ketoacidosis (DKA) is a potentially life-threatening complication of diabetes mellitus. [1] Signs and symptoms may include vomiting, abdominal pain, deep gasping breathing, increased urination, weakness, confusion and occasionally loss of consciousness. [1]
For others, sundowning can show up as confusion. “They will not remember conversations from earlier in the day or what they ate,” Dr. Kobylarz says. That can including wanting to eat dinner ...
Being exposed to bright light during a specific time window at night may heighten the risk of developing type 2 diabetes, a new study suggests. ... Research has found a link between light exposure ...
Diabetes mellitus, also known as type II diabetes, is a disease that affects over three million people in the U.S. per year. This disease affects the glucose levels in the body by causing them to rise higher than normal. In type II diabetic patients, the body develops insulin resistance that initiates an increase in blood glucose levels. [1]