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Pyloric stenosis is a narrowing of the opening from the stomach to the first part of the small intestine (the pylorus). [1] Symptoms include projectile vomiting without the presence of bile. [1] This most often occurs after the baby is fed. [1] The typical age that symptoms become obvious is two to twelve weeks old. [1]
In children, congenital pyloric stenosis / congenital hypertrophic pyloric stenosis may be a cause. A pancreatic pseudocyst can cause gastric compression. Pyloric mucosal diaphragm could be a rare cause. Malignant Tumours of the stomach, including adenocarcinoma (and its linitis plastica variant), lymphoma, and gastrointestinal stromal tumours
The pyloric tit sign is a radiological finding observed during barium studies in cases of hypertrophic pyloric stenosis. [1] It appears as an outpouching on the lesser curvature of the stomach, just proximal to the impression created by the hypertrophied pyloric muscle.
Barium X-ray examinations are useful tools for the study of appearance and function of the parts of the gastrointestinal tract. They are used to diagnose and monitor esophageal reflux, dysphagia, hiatus hernia, strictures, diverticula, pyloric stenosis, gastritis, enteritis, volvulus, varices, ulcers, tumors, and gastrointestinal dysmotility, as well as to detect foreign bodies.
Duodenal atresia is the congenital absence or complete closure of a portion of the lumen of the duodenum.It causes increased levels of amniotic fluid during pregnancy (polyhydramnios) and intestinal obstruction in newborn babies.
[5] [1] Hypertrophic Pyloric stenosis is a gastrointestinal tract defect, most commonly seen in young children, typically in the first few months of life, caused by enlargement of the tissue in the pyloric muscle. [5] [4] [1] This causes the contents of the stomach to be unable to empty leading to pain after eating, electrolyte abnormalities ...
Symptoms include abdominal pain which may come and go, vomiting, abdominal bloating, and bloody stool. [1] It often results in a small bowel obstruction. [1] Other complications may include peritonitis or bowel perforation. [1] The cause in children is typically unknown; in adults a lead point is sometimes present. [1]
The exact definition of "rapid" is somewhat debatable, and different sub-divisions exist, which are based on the time from onset of first hepatic symptoms to onset of encephalopathy. One scheme defines "acute hepatic failure" as the development of encephalopathy within 26 weeks of the onset of any hepatic symptoms.