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Decerebrate posturing is commonly seen in pontine strokes. A patient with decorticate posturing may begin to show decerebrate posturing, or may go from one form of posturing to the other. [1] Progression from decorticate posturing to decerebrate posturing is often indicative of uncal (transtentorial) or tonsilar brain herniation.
In an animal where the cortical areas or the midbrain have been "cut off" from the neural axis, this reflex is hyperactive and the animal will maximally extend all four limbs. This phenomenon is known as decerebrate rigidity. In humans, true decerebrate rigidity is rare since the damage to the brain centers it might be caused by usually are lethal.
Abnormal posturing Opisthotonus or opisthotonos (from Ancient Greek : ὄπισθεν , romanized : opisthen , lit. 'behind' and τόνος , tonos , 'tension') is a state of severe hyperextension and spasticity in which an individual's head, neck and spinal column enter into a complete "bridging" or "arching" position.
Decorticate posturing is a stereotypical posturing in which the patient has arms flexed at the elbow, and arms adducted toward the body, with both legs extended. Decerebrate posturing is a stereotypical posturing in which the legs are similarly extended (stretched), but the arms are also stretched (extended at the elbow). The posturing is ...
The rupture of these vessels leads to linear or flamed shaped hemorrhages. The disrupted brainstem can lead to decorticate posture, respiratory center depression and death. Other possibilities resulting from brain stem distortion include lethargy, slow heart rate, and pupil dilation. [9]
Tonic posturing preceding convulsion has been observed in sports injuries at the moment of impact [2] [3] where extension and flexion of opposite arms occur despite body position or gravity. The fencing response emerges from the separation of tonic posturing from convulsion and refines the tonic posturing phase as an immediate forearm motor ...
Accompanying signs and symptoms may include decerebrate posturing; fixed, dilated pupils; coma or profound stupor; quadriparesis; absent corneal reflex; negative oculocephalic reflex; and obliteration of the gag reflex.
Reye syndrome is a rapidly worsening brain disease. [2] Symptoms of Reye syndrome may include vomiting, personality changes, confusion, seizures, and loss of consciousness. [1]