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Situational syncope is often triggered by urination, swallowing, or coughing. [2] Carotid sinus syncope is due to pressure on the carotid sinus in the neck. [2] The underlying mechanism involves the nervous system slowing the heart rate and dilating blood vessels, resulting in low blood pressure and thus not enough blood flow to the brain. [2]
The carotid sinus baroreceptor can be oversensitive to manual stimulation from the pressure applied at the carotid sinus at the carotid bifurcation. It is a condition known as 'carotid sinus hypersensitivity' (CSH), 'carotid sinus syndrome' or 'carotid sinus syncope', in which manual stimulation causes large changes in heart rate and blood ...
Baroreflex activation therapy is an approach to treating high blood pressure and the symptoms of heart failure.It uses an implanted device to electrically stimulate baroreceptors in the carotid sinus region.
Neurally mediated syncope may also occur when an area in the neck known as the carotid sinus is pressed. [1] A normal response to carotid sinus massage is reduction in blood pressure and slowing of the heart rate. Especially in people with hypersensitive carotid sinus syndrome this response can cause syncope or presyncope. [9]
A vagal maneuver is an action used to stimulate the parasympathetic nervous system by activating the vagus nerve.The vagus nerve is the longest nerve of the autonomic nervous system and helps regulate many critical aspects of human physiology, including heart rate, blood pressure, sweating, and digestion through the release of acetylcholine.
Carotidynia is a syndrome characterized by unilateral (one-sided) tenderness of the carotid artery, near the bifurcation. It was first described in 1927 by Temple Fay. [1] The most common cause of carotidynia may be migraine, and then it is usually self-correcting. Common migraine treatments may help alleviate the carotidynia symptoms.
Atherosclerosis is the most common cause of SSS; [2] all atherosclerotic risk factors are risk factors for SSS. Thoracic outlet syndrome (TOS) increases the risk for SSS. [2] TOS doesn't directly cause SSS, because the site of subclavian artery compression is over the first rib, which is distal to the vertebral artery.
It conveys information from the baroreceptors of the carotid sinus to the vasomotor center in the brainstem (in order to mediate blood pressure homeostasis), and from chemoreceptors of the carotid body [further explanation needed] (mainly conveying information about partial pressures of blood oxygen, and carbon dioxide).