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Familial male-limited precocious puberty, often abbreviated as FMPP, also known as familial sexual precocity or gonadotropin-independent testotoxicosis, [1] is a form of gonadotropin-independent precocious puberty in which boys experience early onset and progression of puberty. [2] Signs of puberty can develop as early as an age of 1 year.
As puberty hits, boys are likely not thinking about children or how their current habits will affect their future fertility. Smoking, drug use, poor diet, obesity — these all can contribute to ...
African-American girls are especially prone to early puberty. [18] Though boys face fewer problems from early puberty than girls do, early puberty is not always positive for boys. Early sexual maturation in boys can be accompanied by increased aggressiveness due to the surge of pubertal hormones. [57]
The human Y chromosome showing the SRY gene which codes for a protein regulating sexual differentiation. Sexual differentiation in humans is the process of development of sex differences in humans. It is defined as the development of phenotypic structures consequent to the action of hormones produced following gonadal determination. [1]
Early puberty may represent a critical window of physiological changes in boys, scientists say
Early adolescence (puberty begins, 10 to 13 years): 4 - 6 ml; Middle adolescence (puberty changes continue, 14 to 17 years): 8 - 10 ml; Late adolescence/young adulthood (18 to 21 years and beyond): 12 - 15 ml; Adulthood: 20 - 25 ml; People with macroorchidism have testicular volume larger than 4 ml before puberty. [11]
Mini-puberty is a transient hormonal activation of the hypothalamic-pituitary-gonadal (HPG) axis that occurs in infants shortly after birth. This period is characterized by a surge in the secretion of gonadotropins (LH and FSH) and sex steroids (testosterone in males and estradiol in females), similar to but less intense than the hormonal changes that occur in puberty during adolescence.
5α-reductase deficiency (5-ARD) – an autosomal recessive condition caused by a mutation of the 5-alpha reductase type 2 gene. It only affects people with Y chromosomes, namely genetic males. People with this condition are fertile, with the ability to father children, but may be raised as females due to ambiguous or feminized genitalia. [18] [19]