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  2. Glucocorticoid remediable aldosteronism - Wikipedia

    en.wikipedia.org/wiki/Glucocorticoid_remediable...

    Aldosterone is increased by blood loss, pregnancy, and possibly by other circumstances such as physical exertion, endotoxin shock, and burns. Aldosterone feedback : [ citation needed ] Feedback by aldosterone concentration itself is of a non-morphological character (that is, other than changes in cell number or structure) and is relatively poor ...

  3. Congenital adrenal hyperplasia - Wikipedia

    en.wikipedia.org/wiki/Congenital_adrenal_hyperplasia

    In 11-hydroxylase deficiency, 11-deoxycorticosterone is produced in such excess that it acts to retain sodium at the expense of potassium. It is this reason that patients with 11-hydroxylase deficiency do not show salt wasting (although sometimes they do in infancy), and instead have hypertension/water retention and sometimes hypokalemia.

  4. Congenital adrenal hyperplasia due to 11β-hydroxylase deficiency

    en.wikipedia.org/wiki/Congenital_adrenal...

    Congenital adrenal hyperplasia due to 11β-hydroxylase deficiency is a form of congenital adrenal hyperplasia (CAH) which produces a higher than normal amount of androgen, [1] resulting from a defect in the gene encoding the enzyme steroid 11β-hydroxylase (11β-OH) which mediates the final step of cortisol synthesis in the adrenal. 11β-OH CAH results in hypertension due to excessive ...

  5. Apparent mineralocorticoid excess syndrome - Wikipedia

    en.wikipedia.org/wiki/Apparent_mineralocorticoid...

    Common symptoms include hypertension, hypokalemia, metabolic alkalosis, and low plasma renin activity. [1] DOC excess syndrome is an excessive secretion of 21-hydroxyprogesterone also called 11-Deoxycorticosterone from adrenal glands and may cause mineralocorticoid hypertension. [4] [5] [6]

  6. Inborn errors of metabolism - Wikipedia

    en.wikipedia.org/wiki/Inborn_errors_of_metabolism

    Inborn errors of metabolism form a large class of genetic diseases involving congenital disorders of enzyme activities. [1] The majority are due to defects of single genes that code for enzymes that facilitate conversion of various substances into others ().

  7. Factor V Leiden - Wikipedia

    en.wikipedia.org/wiki/Factor_V_Leiden

    Factor V Leiden is an autosomal dominant genetic condition that exhibits incomplete penetrance, i.e. not every person who has the mutation develops the disease. The condition results in a factor V variant that cannot be as easily degraded by activated protein C. The gene that codes the protein is referred to as F5.

  8. Gestational Hypertension - Wikipedia

    en.wikipedia.org/wiki/Gestational_hypertension

    Gestational hypertension or pregnancy-induced hypertension (PIH) is the development of new hypertension in a pregnant woman after 20 weeks' gestation without the presence of protein in the urine or other signs of pre-eclampsia. [1] Gestational hypertension is defined as having a blood pressure greater than 140/90 on two occasions at least 6 ...

  9. Bleeding diathesis - Wikipedia

    en.wikipedia.org/wiki/Bleeding_diathesis

    Acute leukemia [5] Transfusion transmitted infection, from blood transfusions that are given as treatment. Hemophilia [7] Adverse reactions to clotting factor treatment. Hemophilia [7] Anemia: Von Willebrand disease [8] Exsanguination (bleeding to death) Von Willebrand disease [8] Acute leukemia [5] Vitamin K deficiency [6] Cerebral hemorrhage