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Nitrogen dioxide is an irritant of the mucous membrane linked with another air pollutant that causes pulmonary diseases such as obstructive lung disease, asthma, chronic obstructive pulmonary disease and sometimes acute exacerbation of COPD and in fatal cases, deaths. [2]
Nitrogen dioxide is poisonous and can be fatal if inhaled in large quantities. [8] Cooking with a gas stove produces nitrogen dioxide which causes poorer indoor air quality. Combustion of gas can lead to increased concentrations of nitrogen dioxide throughout the home environment which is linked to respiratory issues and diseases.
This is a problem, the researchers said, because NO2 exposure over time can make asthma attacks worse and has been linked to affecting children's lung development.
There is strong evidence that NO x respiratory exposure can trigger and exacerbate existing asthma symptoms, and may even lead to the development of asthma over longer periods of time. It has also been associated with heart disease, diabetes, birth outcomes, and all-cause mortality, but these nonrespiratory effects are less well-established. [29]
"This causes recurrent episodes of coughing, wheezing, and chest tightness," says Dr. Laura Conrad, director of the Pediatric Asthma Center at the Children’s Hospital at Montefiore in New York.
[30] [31] Ground-level ozone, sulfur dioxide, nitrogen dioxide and carbon monoxide are especially harmful for senior citizens, children, and people with heart and lung conditions such as emphysema, bronchitis, and asthma. [14] It can inflame breathing passages, decrease the lungs' working capacity, cause shortness of breath, pain when inhaling ...
The role for eNO in other conditions is even less well established compared to asthma. Since asthma can be a cause of chronic coughing (it may even be the sole manifestation, such as in cough-variant asthma), studies have looked at whether eNO can be used in the diagnosis of chronic cough. [18] [19] [20] [21]
Asthma phenotyping and endotyping has emerged as a novel approach to asthma classification inspired by precision medicine which separates the clinical presentations of asthma, or asthma phenotypes, from their underlying causes, or asthma endotypes. The best-supported endotypic distinction is the type 2-high/type 2-low distinction.
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