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  2. File:Control Of The Apoptosis Mecanisms.pdf - Wikipedia

    en.wikipedia.org/wiki/File:Control_Of_The...

    Permission is granted to copy, distribute and/or modify this document under the terms of the GNU Free Documentation License, Version 1.2 or any later version published by the Free Software Foundation; with no Invariant Sections, no Front-Cover Texts, and no Back-Cover Texts.

  3. Cell death - Wikipedia

    en.wikipedia.org/wiki/Cell_death

    Overview of signal transduction pathways involved in apoptosis. Cell death is the event of a biological cell ceasing to carry out its functions. This may be the result of the natural process of old cells dying and being replaced by new ones, as in programmed cell death, or may result from factors such as diseases, localized injury, or the death of the organism of which the cells are part.

  4. Excitotoxicity - Wikipedia

    en.wikipedia.org/wiki/Excitotoxicity

    [19] [20] One of the damaging results of excess calcium in the cytosol is initiating apoptosis through cleaved caspase processing. [20] Another damaging result of excess calcium in the cytosol is the opening of the mitochondrial permeability transition pore, a pore in the membranes of mitochondria that opens when the organelles absorb too much ...

  5. Apoptosis - Wikipedia

    en.wikipedia.org/wiki/Apoptosis

    Unlike necrosis, apoptosis produces cell fragments called apoptotic bodies that phagocytes are able to engulf and remove before the contents of the cell can spill out onto surrounding cells and cause damage to them. [5] Because apoptosis cannot stop once it has begun, it is a highly regulated process.

  6. Mitochondrial outer membrane permeabilization - Wikipedia

    en.wikipedia.org/wiki/Mitochondrial_outer...

    Mitochondrial outer membrane permeabilization (MOMP), also known as the mitochondrial outer membrane permeability, is one of two ways apoptosis (a type of programmed cell death) can be activated. [1] It is part of the intrinsic pathway of apoptosis, also known as the mitochondrial pathway. MOMP is known as the point of no return in apoptosis.

  7. Inhibitor of apoptosis - Wikipedia

    en.wikipedia.org/wiki/Inhibitor_of_apoptosis

    The combinations of the domains in the proteins determine its role in the apoptosis process. Members of the family that inhibit apoptosis include Bcl-2 itself, Bcl-XL, and Bcl-w, which possess all four of the domains. [3] Bcl-2 is the most well known of the anti-apoptotic members, and is classified as an oncogene. Studies have shown that the ...

  8. Fas receptor - Wikipedia

    en.wikipedia.org/wiki/Fas_receptor

    The Fas receptor, also known as Fas, FasR, apoptosis antigen 1 (APO-1 or APT), cluster of differentiation 95 (CD95) or tumor necrosis factor receptor superfamily member 6 (TNFRSF6), is a protein that in humans is encoded by the FAS gene. [5] [6] Fas was first identified using a monoclonal antibody generated by immunizing mice with the FS-7 cell ...

  9. Cellular adaptation - Wikipedia

    en.wikipedia.org/wiki/Cellular_adaptation

    Cellular hypertrophy is an increase in cell size and volume. If enough cells of an organ hypertrophy the whole organ will increase in size. Hypertrophy may involve an increase in intracellular protein as well as cytosol (intracellular fluid) and other cytoplasmic components.