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Citalopram, sold under the brand name Celexa among others, is an antidepressant of the selective serotonin reuptake inhibitor (SSRI) class. [7] [10] It is used to treat major depressive disorder, obsessive compulsive disorder, panic disorder, and social phobia. [7] The antidepressant effects may take one to four weeks to occur. [7]
For adults older than 64, SSRI's seem to reduce the risk of both suicidal behavior. [122] In 2016 a study criticized the effects of the FDA Black Box suicide warning inclusion in the prescription. The authors discussed the suicide rates might increase also as a consequence of the warning. [138]
More serious side effects may include suicidal thoughts in people up to the age of 24 years. [9] It is unclear if use during pregnancy or breastfeeding is safe. [ 10 ] Escitalopram is the ( S )- enantiomer of citalopram (which exists as a racemate ), hence the name es -citalopram.
Bedroom side effects. Anorgasmia (difficulty experiencing climax) is a noted side effect of Lexapro. In women, clinical trials of escitalopram for depression found that roughly two to six percent ...
Citalopram (generic for Celexa) ... only cause ED and other intimate dysfunction issues in 30 percent of patients. Intimate side effects as a result of these medications are often referred to as ...
Celexa – an antidepressant of the SSRI class; Centrax – an anti-anxiety agent; Clozaril – atypical antipsychotic used to treat resistant schizophrenia; Concerta (methylphenidate) – an extended release form of methylphenidate
The most promising therapeutic effect is achieved when the treatment continues for at least six weeks. [36] Underuse or misuse of antidepressants and prescribing inadequate dosages are the most common mistakes physicians make when treating elderly patients for depression. Only 10% to 40% of depressed elderly patients are given medication.
The pharmacology of antidepressants is not entirely clear.. The earliest and probably most widely accepted scientific theory of antidepressant action is the monoamine hypothesis (which can be traced back to the 1950s), which states that depression is due to an imbalance (most often a deficiency) of the monoamine neurotransmitters (namely serotonin, norepinephrine and dopamine). [1]