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The gastrocolic reflex or gastrocolic response is a physiological reflex that controls the motility, or peristalsis, of the gastrointestinal tract following a meal. It involves an increase in motility of the colon consisting primarily of giant migrating contractions, in response to stretch in the stomach following ingestion and byproducts of digestion entering the small intestine. [1]
Stomach pain is a common symptom of functional dyspepsia. Symptoms: Early satiety, heartburn, nausea, postprandial fullness, vomiting, and/or epigastric pain. [1] Complications: Symptoms of anxiety, depression, and somatization. [2] Types: Postprandial distress syndrome and epigastric pain syndrome. [1] Diagnostic method: Rome IV criteria. [1 ...
The chronic form typically presents more gradually with abdominal pain after eating, unintentional weight loss, vomiting, and fear of eating. [ 1 ] [ 2 ] Risk factors for acute intestinal ischemia include atrial fibrillation , heart failure , chronic kidney failure , being prone to forming blood clots , and previous myocardial infarction . [ 2 ]
Lower left abdominal pain can have many causes, ranging from minor to serious, says Andrew Boxer, M.D., gastroenterologist of Gastroenterology Associates of New Jersey. Common causes include ...
A lifestyle change that may help with indigestion is a change in diet, such as a stable and consistent eating schedule and slowing the pace of eating. [31] Additionally, there are studies that support a reduction in the consumption of fats may also alleviate dyspepsia. [31]
Enteritis is inflammation of the small intestine. It is most commonly caused by food or drink contaminated with pathogenic microbes, [1] such as Serratia, but may have other causes such as NSAIDs, radiation therapy as well as autoimmune conditions like coeliac disease. Symptoms include abdominal pain, cramping, diarrhoea, dehydration, and fever ...
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An analysis by University Hospitals Cleveland Medical Center of records from the TriNetX database found that the number of patients diagnosed with gastroparesis after being prescribed a GLP-1 receptor agonist (0.1% of the patients) was 250% greater than the number of patients diagnosed with gastroparesis who did not take a GLP-1 medication (0.04%).