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  2. List of herbs with known adverse effects - Wikipedia

    en.wikipedia.org/wiki/List_of_herbs_with_known...

    Kava: kava-kava Piper methysticum: Sedatives, sleeping pills, antipsychotics, alcohol [15] Milkvetch: Astragalus: Astragalus may interact with medications that suppress the immune system, such as cyclophosphamide. [24] It may also affect blood sugar levels and blood pressure. Pineapple enzyme Ananas comosus: Bromelain

  3. The 7 Most Effective Natural Sleep Aids for Your Best Sleep ...

    www.aol.com/lifestyle/7-most-effective-natural...

    In sleep medicine, we use very small doses of melatonin multiple hours before bed to help gradually shift the body’s clock (circadian rhythm) in people with delayed sleep phase disorder, jet lag ...

  4. Relaxation drink - Wikipedia

    en.wikipedia.org/wiki/Relaxation_drink

    Kava roots are well known in the Pacific Islands and are mostly grown in Tonga and Fiji. Kava is known to be used for social rituals and celebrations. Melatonin is another major ingredient found in relaxation drinks which also carry some controversy due to the negative effects of long-term use. Relaxation drinks have been known to contain other ...

  5. Kava - Wikipedia

    en.wikipedia.org/wiki/Kava

    Kava or kava kava (Piper methysticum: Latin 'pepper' and Latinized Greek 'intoxicating') is a plant in the pepper family, native to the Pacific Islands. [1] The name kava is from Tongan and Marquesan, meaning 'bitter.’ [1] Other names for kava include ʻawa (), [2] ʻava (), yaqona or yagona (), [3] sakau (), [4] seka (), [5] and malok or malogu (parts of Vanuatu). [6]

  6. 5 Best CBD Products for Sleep - AOL

    www.aol.com/lifestyle/5-best-cbd-products-sleep...

    The National Sleep Foundation recommends that healthy adults get between seven and nine hours a night. The scientific jury is still out when it comes to sleep in general and sleep conditions like ...

  7. Kavain - Wikipedia

    en.wikipedia.org/wiki/Kavain

    Kavain has anticonvulsive properties, attenuating vascular smooth muscle contraction through interactions with voltage-dependent Na + and Ca 2+ channels. [1] How this effect is mediated, and to what extent this mechanism is involved in the anxiolytic and analgesic effects of kavalactones on the central nervous system, is unknown.

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