Search results
Results from the WOW.Com Content Network
Echopraxia is a typical symptom of Tourette syndrome but causes are not well elucidated. [1]Frontal lobe animation. One theoretical cause subject to ongoing debate surrounds the role of the mirror neuron system (MNS), a group of neurons in the inferior frontal gyrus (F5 region) of the brain that may influence imitative behaviors, [1] but no widely accepted neural or computational models have ...
This lower-than-normal B12 profile persisted throughout life in the brain tissues of patients with autism. These deficiencies are not visible by conventional blood sampling. [104] [105] As for the classic deficiency of vitamin B12, it would affect up to 40% of the population, its prevalence has not yet been studied in autism spectrum disorders ...
The imprinted brain hypothesis is an unsubstantiated hypothesis in evolutionary psychology regarding the causes of autism spectrum and schizophrenia spectrum disorders, first presented by Bernard Crespi and Christopher Badcock in 2008.
Mind-blindness is defined as a state where the ToM has not been developed in an individual. [1] According to the theory, non-autistic people can make automatic interpretations of events taking into consideration the mental states of people, their desires, and beliefs.
There is no scientific consensus on its classification as a personality trait, medical symptom, or mental disorder. [7] [8] Alexithymia occurs in approximately 10% of the population and often co-occurs with various mental or neurodevelopmental disorders. [9] It is present in 50% to 85% of individuals with autism spectrum disorder (ASD). [10]
The weak central coherence theory (WCC), also called the central coherence theory (CC), suggests that a specific perceptual-cognitive style, loosely described as a limited ability to understand context or to "see the big picture", underlies the central issue in autism and related autism spectrum disorder. Autism is a neurodevelopmental disorder ...
The amygdala, cerebellum, and many other brain regions have been implicated in autism. [15]Unlike some brain disorders which have clear molecular hallmarks that can be observed in every affected individual, such as Alzheimer's disease or Parkinson's disease, autism does not have a unifying mechanism at the molecular, cellular, or systems level.
Rett syndrome brain samples and autism brain samples show immaturity of dendrite spines and reduction of cell-body size due to errors in coupled regulation between MECP2 and EGR2. [62] However, because of the multigene involvement in autism, the MECP2 gene has only been identified as a vulnerability factor in autism. [ 63 ]