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The esophageal veins drain blood from the esophagus to the azygos vein, in the thorax, and to the inferior thyroid vein in the neck. It also drains, although with less significance, to the hemiazygos vein , posterior intercostal vein and bronchial veins .
Esophageal varices seven days after banding, showing ulceration at the site of banding. The upper two thirds of the esophagus are drained via the esophageal veins, which carry deoxygenated blood from the esophagus to the azygos vein, which in turn drains directly into the superior vena cava. These veins have no part in the development of ...
Blue rubber bleb nevus syndrome is a rare disorder that consists mainly of abnormal blood vessels affecting the skin or internal organs – usually the gastrointestinal tract. [1] The disease is characterized by the presence of fluid-filled blisters ( blebs ) as visible, circumscribed, chronic lesions ( nevi ).
Typically this occurs due to portal hypertension which shunts venous blood from the portal system through the portosystemic anastomosis present at this site into the systemic venous system. [2] [3] This can also occur in the esophagus, causing esophageal varices, and at the level of the umbilicus, causing caput medusae. [4]
Blebs can form in a number of tissues by different pathologies, including frostbite and can "appear and disappear within a short time interval". In pathology, pulmonary blebs are small subpleural thin-walled air-containing spaces, not larger than 1-2 cm in diameter, found by the upper lobe of the lung, between the lung and the visceral pleura .
Some people also experience a sensation known as globus esophagus, where it feels as if a ball is lodged in the lower part of the esophagus. The following are additional diseases and conditions that affect the esophagus: Achalasia [1] Acute esophageal necrosis; Barrett's esophagus; Boerhaave syndrome; Caustic injury to the esophagus; Chagas disease
The esophageal branch of the left gastric vein drains into the azygos vein. In cases of portal hypertension, this communication allows for blood to bypass the portal vein and reach systemic circulation. As a result of this anastomosis, development of esophageal and paraesophageal varices is possible. [3]
The symptoms vary from the severity of the disorder. The most classic sign of AEN is the dark pigmentation of esophageal mucosa in an upper endoscopy, usually viewed as an ulcer or as an infectious disease. [6] Necrosis can be found mostly between the three distals of the esophagus, but stops abruptly at the gastroesophageal junction. [2]