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Cutaneous manifestations of COVID-19 are characteristic signs or symptoms of the Coronavirus disease 2019 that occur in the skin. The American Academy of Dermatology reports that skin lesions such as morbilliform (measles-like rashes, 22%), pernio (capillary damage, 18%), urticaria (hives, 16%), macular erythema (rose-colored rash, 13%), vesicular purpura (purplish discolouration, 11% ...
The COVID-19 pandemic in Indonesia is part of the worldwide pandemic of coronavirus disease 2019 (COVID-19) caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). It was confirmed to have spread to Indonesia on 2 March 2020, after a dance instructor and her mother tested positive for the virus. Both were infected from a ...
Fever in the first week of a COVID-19 infection is part of the body's natural immune response; however in severe cases, if the infections develop into a cytokine storm the fever is counterproductive. As of September 2020, little research had focused on relating fever intensity to outcomes.
The COVID-19 pandemic in Southeast Asia is part of the ongoing worldwide pandemic of coronavirus disease 2019 (COVID-19) caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). It was confirmed to have spread to Southeast Asia on 13 January 2020, when a 61-year-old woman from Wuhan tested positive in Thailand , making it the ...
The COVID-19 pandemic in Turkey is part of the ongoing pandemic of coronavirus disease 2019 (COVID-19) caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). The first case in Turkey was recorded on 11 March, when a local returned home [ note 1 ] from a trip to Europe. [ 302 ]
The first cases of COVID-19 were confirmed in Indonesia on 2 March 2020, when two residents of Depok, West Java tested positive for the virus. [4] On 15 March, with 117 confirmed cases, President Joko Widodo had called for Indonesians to exercise social distancing measures, with some regional leaders in Jakarta, Banten and West Java had already closed down schools and places of gathering. [5]
Virus infections start when viral particles bind to host surface cellular receptors. [158] Protein modeling experiments on the spike protein of the virus soon suggested that SARS‑CoV‑2 has sufficient affinity to the receptor angiotensin converting enzyme 2 (ACE2) on human cells to use them as a mechanism of cell entry. [159]
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