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Malathion is an acetylcholinesterase inhibitor, a diverse family of chemicals.Upon uptake into the target organism, it binds irreversibly to the serine residue in the active catalytic site of the cholinesterase enzyme.
Azinphos-methyl is a neurotoxin derived from nerve agents developed during World War II. [4] It was first registered in the US in 1959 as an insecticide and is also used as active ingredient in organophosphate (OP) pesticides. [6]
Neurotoxicity is a form of toxicity in which a biological, chemical, or physical agent produces an adverse effect on the structure or function of the central and/or peripheral nervous system. [1] It occurs when exposure to a substance – specifically, a neurotoxin or neurotoxicant – alters the normal activity of the nervous system in such a ...
Neurotoxic effects have also been linked to poisoning with OP pesticides causing four neurotoxic effects in humans: cholinergic syndrome, intermediate syndrome, organophosphate-induced delayed polyneuropathy (OPIDP), and chronic organophosphate-induced neuropsychiatric disorder (COPIND). These syndromes result after acute and chronic exposure ...
One of the key uses for the compound is in production of the pesticide Malathion. [5] It has also been used medically as a chemical depletory of glutathione. [6] It has been studied extensively with regard to renal function. [7] Other medical uses include treatment of breast cancer and its monitoring with Positron Emission Tomography. [8]
Malathion: Malathion USB, ~ EC, Cythion, maldison, mercaptothion Organophosphate >8 fl oz/acre (58 L/km 2) ⇒ 5.5 days Malathion is highly toxic to bees and other beneficial insects, some fish, and other aquatic life. Malathion is moderately toxic to other fish and birds, and is considered low in toxicity to mammals. [40] Highly toxic ...
Human Brain Project (EU) – 1 billion euro, 10-year project to simulate the human brain with supercomputers. BigBrain A high-resolution 3D atlas of the human brain created as part of the HBP. Human Connectome Project – 2009 NIH $30 million project to build a network map of the human brain, including structural (anatomical) and functional ...
Beta activity is increased when movement has to be resisted or voluntarily suppressed. [8] The artificial induction of increased beta waves over the motor cortex by a form of electrical stimulation called Transcranial alternating-current stimulation consistent with its link to isotonic contraction produces a slowing of motor movements.