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Rodent mite dermatitis (also known as rat mite dermatitis) is an often unrecognized ectoparasitosis occurring after human contact with haematophagous mesostigmatid mites that infest rodents, such as house mice, [1] rats [2] and hamsters. [3]
Rarely, the disease may be spread by blood transfusion or other organ transplant. [3] It is not otherwise spread between people. [3] The parasite is known to reproduce sexually only in the cat family. [9] However, it can infect most types of warm-blooded animals, including humans. [9]
Ornithonyssus bacoti (also known as the tropical rat mite and formerly called Liponyssus bacoti) is a hematophagous parasite. [1] It feeds on blood and serum from many hosts. [2] [3] O. bacoti can be found and cause disease on rats and wild rodents most commonly, but also small mammals and humans when other hosts are scarce.
Rat-bite fever (RBF) is an acute, febrile human illness caused by bacteria transmitted by rodents, in most cases, which is passed from rodent to human by the rodent's urine or mucous secretions. Alternative names for rat-bite fever include streptobacillary fever, streptobacillosis, spirillary fever, bogger, and epidemic arthritic erythema.
In human adults, the tapeworm is more of a nuisance than a health problem, but in small children, many H. nana worms can be dangerous. Usually, the larvae of this tapeworm cause the most problem in children; they burrow into the walls of the intestine, and if enough tapeworms are present in the child, severe damage can be inflicted.
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Rodents, especially rats, are definitive hosts and natural reservoirs of H. microstoma. As the definitive host (rats) eats an infected arthropod, cysticercoids present in the body cavity transform into the adult worm. Juvenile worms establish in the bile duct of mice after approximately 3 days movement within the upper gastrointestinal tract.
Piebaldism has been documented to occur in all races, and is found in nearly every species of mammal. The condition is very common in mice, rabbits, dogs, sheep, deer, cattle and horses—where selective breeding has increased the incidence of the mutation—but occurs among chimpanzees and other primates only as rarely as among humans.