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Oncogene received a 2023 impact factor of 6.9 and received Journal Citation Reports rankings of 18th out of 191 in the category Genetics & Heredity, 29th out of 205 in the category Cell Biology, 32nd out of 313 in the category Biochemistry & Molecular Biology, and 43rd out of 322 journals in the category Oncology. [3]
Oncogenesis is a peer-reviewed open access medical journal covering the molecular biology of cancer. It was established in 2012 by Douglas R. Green as a sister journal to Oncogene, of which Green was then editor-in-chief. [1] New articles are published exclusively online by Springer Nature on a weekly basis. [2]
Database Institute / Organization Alteration Types Primary Source [t 1] Processed Data [t 2] Organisms Cell lines [t 3] Public Data [t 4] Restricted Data [t 5]; The BioExpress® Oncology Suite from Ocimum Bio Solutions contains gene expression data from primary, metastatic, and benign tumor samples, and normal samples, including matched adjacent controls.
Protein c-Fos is a proto-oncogene that is the human homolog of the retroviral oncogene v-fos. [5] It is encoded in humans by the FOS gene. It was first discovered in rat fibroblasts as the transforming gene of the FBJ MSV (Finkel–Biskis–Jinkins murine osteogenic sarcoma virus) (Curran and Tech, 1982).
This gene is the cellular homolog of the Yamaguchi sarcoma virus oncogene. The encoded protein has tyrosine kinase activity and belongs to the src family . This gene lies in close proximity to thymidylate synthase gene on chromosome 18 , and a corresponding pseudogene has been found on chromosome 22.
n/a Ensembl ENSG00000249859 n/a UniProt n a n/a RefSeq (mRNA) n/a n/a RefSeq (protein) n/a n/a Location (UCSC) Chr 8: 127.79 – 128.19 Mb n/a PubMed search n/a Wikidata View/Edit Human Pvt1 oncogene (non-protein coding), also known as PVT1 or Plasmacytoma Variant Translocation 1 is a long non-coding RNA gene. In mice, this gene was identified as a breakpoint site in chromosome 6 ; 15 ...
Oncogenomics is a sub-field of genomics that characterizes cancer-associated genes.It focuses on genomic, epigenomic and transcript alterations in cancer. Cancer is a genetic disease caused by accumulation of DNA mutations and epigenetic alterations leading to unrestrained cell proliferation and neoplasm formation.
Journal of Cancer Research and Therapeutics; Journal of Cancer Survivorship; Journal of Carcinogenesis; Journal of Cytology; Journal of Environmental Science and Health, Part C; Journal of Experimental & Clinical Cancer Research; Journal of Medical Imaging and Radiation Oncology; Journal of Pediatric Oncology Nursing; Journal of Surgical Oncology