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(H53.0) Amblyopia (lazy eye) — poor or blurry vision due to either no transmission or poor transmission of the visual image to the brain (H53.0) Leber's congenital amaurosis — genetic disorder; appears at birth, characterised by sluggish or no pupillary responses
Amblyopia, also called lazy eye, is a disorder of sight in which the brain fails to fully process input from one eye and over time favors the other eye. [1] It results in decreased vision in an eye that typically appears normal in other aspects. [ 1 ]
An ocular manifestation of a systemic disease is an eye condition that directly or indirectly results from a disease process in another part of the body. There are many diseases known to cause ocular or visual changes.
This is a very rare tumor, since only about 1 in 35,000 to 40,000 people have VHL, of whom about 10% have endolymphatic sac tumors. Patients usually present in the 4th to 5th decades without an gender predilection. The tumor involves the endolymphatic sac, a portion of the intraosseous inner ear of the posterior petrous bone. [1] [5]
The brain can eliminate double vision by ignoring all or part of the image of one of the eyes. The area of a person's visual field that is suppressed is called the suppression scotoma (with a scotoma meaning, more generally, an area of partial alteration in the visual field). Suppression can lead to amblyopia.
Common causes of scotomas include demyelinating disease such as multiple sclerosis (retrobulbar neuritis), damage to nerve fiber layer in the retina (seen as cotton wool spots [3]) due to hypertension, toxic substances such as methyl alcohol, ethambutol and quinine, nutritional deficiencies, vascular blockages either in the retina or in the optic nerve, stroke or other brain injury, and ...
1.5.4 Diffuse glioneuronal tumor with oligodendroglioma-like features and nuclear clusters 1.5.5 Papillary glioneuronal tumor 1.5.6 Rosette-forming glioneuronal tumor 1.5.7 Myxoid glioneuronal tumor 1.5.8 Diffuse leptomeningeal glioneuronal tumor 1.5.9 Gangliocytoma 1.5.10 Multinodular and vacuolating neuronal tumor
The most common symptoms of acquired and transient cortical blindness include: A complete loss of visual sensation and of vision [4] Preservation/sparing of the abilities to perceive light and/or moving, but not static objects (Riddoch syndrome) [2] A lack of visual fixation and tracking [4] Denial of visual loss (Anton–Babinski syndrome)