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Alcohol-related brain damage [1] [2] alters both the structure and function of the brain as a result of the direct neurotoxic effects of alcohol intoxication or acute alcohol withdrawal. Increased alcohol intake is associated with damage to brain regions including the frontal lobe , [ 3 ] limbic system , and cerebellum , [ 4 ] with widespread ...
5. Their bowls are too close together. This sounds silly, but cats don’t like their food and water close together. That’s because, in the wild, they wouldn’t want to contaminate their clean ...
Toxicity and death may occur after drinking even in a small amount [1] as ethylene glycol is more toxic than other diols. Ethylene glycol is a colorless, odorless, sweet liquid, commonly found in antifreeze. [1] It may be drunk accidentally or intentionally in a suicide attempt. [2]
These adverse effects are believed to be due to the neurotoxic effects of repeated withdrawal from alcohol on aberrant neuronal plasticity and cortical damage. Repeated periods of acute intoxication followed by acute detoxification has profound effects on the brain and is associated with an increased risk of seizures as well as cognitive deficits.
Additionally, drinking alcohol can put a dent in a person's nutrition, preventing the body from absorbing folic acid, which is critical to all our cells. It can also block the uptake of important ...
Alcohol hallucinosis is a rather uncommon alcohol-induced psychotic disorder almost exclusively seen in chronic alcoholics who have many consecutive years of severe and heavy drinking during their lifetime. [3] Alcoholic hallucinosis develops about 12 to 24 hours after the heavy drinking stops suddenly, and can last for days.
Many report a drop in weight after removing the empty calories consumed when drinking. Alcohol, like other foods and drinks that are high in sugar, can not only add unwanted pounds, but may also ...
Aβ was found manipulating the level of nicotine in the brain along with the MAP kinase, another signaling receptor, to cause cell death. Another chemical in the brain that Aβ regulates is JNK; this chemical halts the extracellular signal-regulated kinases (ERK) pathway, which normally functions as memory control in the brain. As a result ...