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Patients who developed antibodies against HBsAg (anti-HBsAg seroconversion) are usually considered non-infectious. HBsAg detection by immunoassay is used in blood screening , to establish a diagnosis of hepatitis B infection in the clinical setting (in combination with other disease markers) and to monitor antiviral treatment .
Anti-HBs concentration falls as the infection resolves but does not serorevert completely, and anti-HBs IgG remains positive for years as a sign of immunity. [65] Hepatitis B e-antigen (HBeAg) is a sign of current infectivity. An individual who is seropositive for HBeAg can infect others. [67]
If the host is able to clear the infection, eventually the HBsAg will become undetectable and will be followed by IgG antibodies to the hepatitis B surface antigen and core antigen (anti-HBs and anti HBc IgG). [39] The time between the removal of the HBsAg and the appearance of anti-HBs is called the window period. A person negative for HBsAg ...
Viral hepatitis is primarily diagnosed through blood tests for levels of viral antigens (such as the hepatitis B surface or core antigen), anti-viral antibodies (such as the anti-hepatitis B surface antibody or anti-hepatitis A antibody), or viral DNA/RNA. [17] [32] In early infection (i.e. within 1 week), IgM antibodies are found in the blood ...
Hepatitis B is one of a few known non-retroviral viruses which use reverse transcription as a part of its replication process. Attachment The virus gains entry into the cell by binding to receptors on the surface of the cell and entering it by endocytosis mediated by either clathrin or caveolin-1 . [ 51 ]
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An anti-Hbs antibody level above 100 mIU/ml is deemed adequate and occurs in about 85–90% of individuals. [33] An antibody level between 10 and 100 mIU/ml is considered a poor response, and these people should receive a single booster vaccination at this time, but do not need further retesting. [ 33 ]