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Surgical third nerve palsy is characterized by a sudden onset of unilateral ptosis and an enlarged or sluggish pupil to the light. In this case, imaging tests such as CTs or MRIs should be considered. Medical third nerve palsy, contrary to surgical third nerve palsy, usually does not affect the pupil and tends to improve in several weeks slowly.
A relative afferent pupillary defect (RAPD), also known as a Marcus Gunn pupil (after Robert Marcus Gunn), is a medical sign observed during the swinging-flashlight test [1] whereupon the patient's pupils excessively dilate when a bright light is swung from the unaffected eye to the affected eye. The affected eye still senses the light and ...
The signs and symptoms occur on the same side (ipsilateral) as it is a lesion of the sympathetic trunk. It is characterized by miosis (a constricted pupil), partial ptosis (a weak, droopy eyelid), apparent anhidrosis (decreased sweating), with apparent enophthalmos (inset eyeball). [2]
Hutchinson's pupil is a clinical sign in which the pupil on the side of an intracranial mass lesion is dilated and unreactive to light, [1] due to compression of the oculomotor nerve on that side. The sign is named after Sir Jonathan Hutchinson. These can be due to concussion injury to the brain and is associated with subdural haemorrhage and ...
After the underlying cause is addressed, vision is not restored right away, as the mechanism also involves the brain. [6] [7] Amblyopia can be difficult to detect, so vision testing is recommended for all children around the ages of four to five. [2] Early detection improves treatment success. [2] Glasses may be all the treatment needed for ...
Closed-head injuries are caused primarily by vehicular accidents, falls, acts of violence, and sports injuries. [4] Falls account for 35.2% of brain injuries in the United States, with rates highest for children ages 0–4 years and adults ages 75 years and older. [3] Head injuries are more common in men than women across every age group. [3]
Second-impact syndrome (SIS) occurs when the brain swells rapidly, and catastrophically, after a person has a second concussion before symptoms from an earlier one have subsided. This second blow may occur minutes, days, or weeks after an initial concussion, [ 1 ] and even the mildest grade of concussion can lead to second impact syndrome. [ 2 ]
Adie's pupil is supersensitive to ACh so a muscarinic agonist (e.g. pilocarpine) whose dose would not be able to cause pupillary constriction in a normal patient, would cause it in a patient with Adie's Syndrome. The circuitry for the pupillary constriction does not descend below the upper midbrain, henceforth impaired pupillary constriction is ...