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Ventral to each of the magnocellular and parvocellular layers lie the koniocellular layers, which differ in thickness. In macaques there are two magnocellular and four parvocellular layers, and accordingly six konicellular layers. K1, the layer ventral to M1, is the largest. K2, K3 and K4 are thinner but nonetheless substantial bands of neurons.
Vitamin K 2, the main storage form in animals, has several subtypes, which differ in isoprenoid chain length. These vitamin K 2 homologues are called menaquinones, and are characterized by the number of isoprenoid residues in their side chains.
The entorhinal cortex (EC) is an area of the brain's allocortex, located in the medial temporal lobe, whose functions include being a widespread network hub for memory, navigation, and the perception of time. [1] The EC is the main interface between the hippocampus and neocortex.
Brain at the U.S. National Library of Medicine Medical Subject Headings (MeSH) (view tree for regions of the brain) BrainMaps.org; BrainInfo (University of Washington) "Brain Anatomy and How the Brain Works". Johns Hopkins Medicine. 14 July 2021. "Brain Map". Queensland Health. 12 July 2022.
The brain is the central organ of the human nervous system, and with the spinal cord, comprises the central nervous system. It consists of the cerebrum, the brainstem and the cerebellum. The brain controls most of the activities of the body, processing, integrating, and coordinating the information it receives from the sensory nervous system ...
Vitamin K2 is lauded for a number of important health benefits. But taking too much can be dangerous, health experts say. Vitamin K2 is essential to your health.
The retrosplenial cortex (RSC) is a cortical area in the brain comprising Brodmann areas 29 and 30. [1] It is secondary association cortex, making connections with numerous other brain regions. The region's name refers to its anatomical location immediately behind the splenium of the corpus callosum in primates, although in rodents it is ...
The resulting decrease in acetylcholine in the brain is thought to contribute to the decline in mental function of affected patients. [ 3 ] [ 2 ] For this reason, most currently available pharmacological treatments for dementia focus on compensating for faltering function of the nucleus basalis through artificially increasing acetylcholine levels.