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Digestive enzyme supplements are a popular go-to for gastric ... from being digested by the acid contained in, and secreted by, the stomach,” Cohen said, so “the enzyme can safely get to the ...
Some antacids also inhibit pepsin, an enzyme that can damage the esophagus in acid reflux. [2] [13] Antacids do not directly inhibit acid secretion, and thus are distinct from acid-reducing drugs like H 2-receptor antagonists or proton pump inhibitors. [4] Antacids do not kill the bacteria Helicobacter pylori, which causes most ulcers. [4]
Bile acid malabsorption (BAM), known also as bile acid diarrhea, is a cause of several gut-related problems, the main one being chronic diarrhea.It has also been called bile acid-induced diarrhea, cholerheic or choleretic enteropathy, bile salt diarrhea or bile salt malabsorption.
Acid peptic diseases, such as peptic ulcers, Zollinger-Ellison syndrome, and gastroesophageal reflux disease, are caused by distinct but overlapping pathogenic mechanisms involving acid effects on mucosal defense. Acid reflux damages the esophageal mucosa and may also cause laryngeal tissue injury, leading to the development of pulmonary ...
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Gastroesophageal reflux disease (GERD) or gastro-oesophageal reflux disease (GORD) is a chronic upper gastrointestinal disease in which stomach content persistently and regularly flows up into the esophagus, resulting in symptoms and/or complications.
Stomach diseases include gastritis, gastroparesis, Crohn's disease and various cancers. [1]The stomach is an important organ in the body. It plays a vital role in digestion of foods, releases various enzymes and also protects the lower intestine from harmful organisms.
A final stage enzyme in the urea cycle is arginase, an enzyme that is crucial to the pathogenesis of H. pylori. Arginase produces ornithine and urea, which the enzyme urease breaks down into carbonic acid and ammonia. Urease is the bacterium’s most abundant protein, accounting for 10–15% of the bacterium's total protein content.
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