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SARS was a relatively rare disease; at the end of the epidemic in June 2003, the incidence was 8,422 cases with a case fatality rate (CFR) of 11%. [5] No cases of SARS-CoV-1 have been reported worldwide since 2004. [6] In December 2019, a second strain of SARS-CoV was identified: SARS-CoV-2. [7]
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SARS-related coronavirus is a member of the genus Betacoronavirus (group 2) and monotypic of the subgenus Sarbecovirus (subgroup B). [13] Sarbecoviruses, unlike embecoviruses or alphacoronaviruses, have only one papain-like proteinase (PLpro) instead of two in the open reading frame ORF1ab. [14]
The template provides data on the COVID-19 pandemic, including cases, deaths, and recoveries.
SARS-CoV-2 is the seventh known coronavirus to infect people, after 229E, NL63, OC43, HKU1, MERS-CoV, and the original SARS-CoV. [105] Like the SARS-related coronavirus implicated in the 2003 SARS outbreak, SARS‑CoV‑2 is a member of the subgenus Sarbecovirus (beta-CoV lineage B). [106] [107] Coronaviruses undergo frequent recombination. [108]
Another common finding in SARS patients is a decrease in the number of lymphocytes circulating in the blood. [14] In the SARS outbreak of 2003, about 9% of patients with confirmed SARS-CoV-1 infection died. [15] The mortality rate was much higher for those over 60 years old, with mortality rates approaching 50% for this subset of patients. [15]
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The Weekly Epidemiological Record was first published by a group of epidemiologists based in the Health Office of the League of Nations, in Geneva, on 1 April 1926, 20 years before the constitution of the World Health Organization was signed at the International Health Conference in New York.