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The symptoms of blood agent poisoning depend on concentration and duration. Cyanide-based blood agents irritate the eyes and the respiratory tract, while arsine is nonirritating. [2] Hydrogen cyanide has a faint, bitter, almond odor that only about half of all people can smell. Arsine has a very faint garlic odor detectable only at greater than ...
Amygdalin (from Ancient Greek: ἀμυγδαλή amygdalē 'almond') is a naturally occurring chemical compound found in many plants, most notably in the seeds (kernels, pips or stones) of apricots, bitter almonds, apples, peaches, cherries and plums, and in the roots of manioc.
The acute oral lethal dose of cyanide for adult humans is reported to be 0.5–3.5 mg/kg (0.2–1.6 mg/lb) of body weight (approximately 50 bitter almonds), so that for children consuming 5–10 bitter almonds may be fatal. [21] Symptoms of eating such almonds include vertigo and other typical cyanide poisoning effects. [55]
Strychnine poisoning is poisoning induced by strychnine. It can be fatal to humans and other animals and can occur by inhalation, swallowing or absorption through eyes or mouth. It produces some of the most dramatic and painful symptoms of any known toxic reaction, making it quite noticeable and a common choice for assassinations and poison ...
Common symptoms are often flulike and include headache, dizziness, upset stomach, chest pain or confusion, but highly concentrated levels of CO can cause a person to pass out without feeling symptoms.
Hydrogen cyanide is a linear molecule, with a triple bond between carbon and nitrogen.The tautomer of HCN is HNC, hydrogen isocyanide. [citation needed]HCN has a faint bitter almond-like odor that some people are unable to detect owing to a recessive genetic trait. [12]
Symptoms usually show up two to six hours after eating or contact with the animal product, according to the CDC, and they include: Hives or itchy rash. Nausea or vomiting. Severe stomach pain.
What are the symptoms of carbon monoxide poisoning? In high concentrations, it can be deadly. The acute effects arise from carboxyhemoglobin formation in the blood, which hampers oxygen absorption.