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The superior cerebellar veins pass partly forward and medialward, across the superior cerebellar vermis.They end in the straight sinus, [1] and the internal cerebral veins, partly lateralward to the transverse and superior petrosal sinuses.
The venous drainage of the cerebrum can be separated into two subdivisions: superficial and deep. The superficial system. The superficial system is composed of dural venous sinuses, sinuses (channels) within the dura mater. The dural sinuses are therefore located on the surface of the cerebrum.
CSF returns to the vascular system by entering the dural venous sinuses via arachnoid granulations. [2] These are outpouchings of the arachnoid mater into the venous sinuses around the brain, with valves to ensure one-way drainage. [2] This occurs because of a pressure difference between the arachnoid mater and venous sinuses. [3]
These sinuses play a crucial role in cerebral venous drainage. A dural venous sinus, in human anatomy, is any of the channels of a branching complex sinus network that lies between layers of the dura mater, the outermost covering of the brain, and functions to collect oxygen-depleted blood. Unlike veins, these sinuses possess no muscular coat.
The pathophysiology of cranial venous outflow obstruction involves the disruption of normal venous drainage from the brain. Cerebral veins play a crucial role in draining brain interstitial fluid (ISF), and their significance has been linked in various neurological conditions. [1] It can be caused by extrinsic or intrinsic anomalies. [7]
A developmental venous anomaly (DVA, formerly known as venous angioma) is a congenital variant of the cerebral venous drainage. On imaging it is seen as a number of small deep parenchymal veins converging toward a larger collecting vein.
The primary goal in cerebral edema is to optimize and regulate cerebral perfusion, oxygenation, and venous drainage, decrease cerebral metabolic demands, and to stabilize the osmolality pressure gradient between the brain and the surrounding vasculature. [3]
Intracerebral hemorrhage into the cerebellum may cause ataxia, vertigo, incoordination of limbs and vomiting. [7] Some cases of cerebellar hemorrhage lead to blockage of the fourth ventricle with subsequent impairment of drainage of cerebrospinal fluid from the brain. [7]