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The frontal eye fields (FEF) are a region located in the frontal cortex, more specifically in Brodmann area 8 or BA8, [1] of the primate brain. In humans, it can be more accurately said to lie in a region around the intersection of the middle frontal gyrus with the precentral gyrus , consisting of a frontal and parietal portion. [ 2 ]
Brodmann area 8, or BA8, is part of the frontal cortex in the human brain. Situated just anterior to the premotor cortex , it includes the frontal eye fields (so-named because they are believed to play an important role in the control of eye movements). Damage to this area, by stroke, trauma or infection, causes tonic deviation of the eyes ...
If any part of the brain that controls eye movement becomes damaged, then OMA may develop. [2] One of the potential causes is bifrontal hemorrhages. In this case, OMA is associated with bilateral lesions of the frontal eye fields (FEF), located in the caudal middle frontal gyrus. The FEF control voluntary eye movements, including saccades ...
Frontal eye field. The frontal eye field is a posterior part of the middle frontal gyrus and is involved in the control of saccadic, contralateral and conjugate eye movement. This area receives its main inputs from both the occipital cortex and dorsal thalamus. [6] Broca's area
In a 2006 study, patients with left prefrontal lesions on the SFG exhibited poorer results on working memory tasks than the control group. Mapping showed the lateral and posterior portions of the SFG contributed the most to the working memory deficit (mostly in Brodmann area 8 in front of the frontal eye field). This study suggests the SFG is ...
Pontine lesions can typically be distinguished from supranuclear lesions in the frontal lobe based on clinical neurologic findings. Gaze palsies secondary to frontal lobe lesions can be temporarily relieved with rapid, passive horizontal head rotation, which also directly stimulates the sixth nerve nucleus through the vestibuloocular reflex .
In the classical sense, expressive aphasia is the result of injury to Broca's area; it is often the case that lesions in specific brain areas cause specific, dissociable symptoms, [31] although case studies show there is not always a one-to-one mapping between lesion location and aphasic symptoms. [29]
Certain parts of the cerebral cortex (including the frontal eye fields), as in stroke. Toxic envenomation by mambas, taipans, and kraits. Thiamine deficiency can cause ophthalmoparesis in susceptible persons; this is part of the syndrome called Wernicke encephalopathy. The causal pathway by which this occurs is unknown.