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2. Hemochromatosis type 4 - Wikipedia

   en.wikipedia.org/wiki/Hemochromatosis_type_4

   Diagnosis is based upon identification of symptoms, medical history, family history, and laboratory tests. Blood tests may show high levels of ferritin and low, normal, or high levels of transferrin saturation, depending on the form of hemochromatosis. The diagnosis must be confirmed by genetic testing for SLC40A1 mutations. [14]

3. Hereditary haemochromatosis - Wikipedia

   en.wikipedia.org/wiki/Hereditary_haemochromatosis

   Normally, HFE facilitates the binding of transferrin, which is iron's carrier protein in the blood. Transferrin levels are typically elevated at times of iron depletion (low ferritin stimulates the release of transferrin from the liver). When transferrin is high, HFE works to increase the intestinal release of iron into the blood.

4. HFE H63D gene mutation - Wikipedia

   en.wikipedia.org/wiki/HFE_H63D_gene_mutation

   The typical constellation of findings is indicative: The patients show a postprandial non-responsive and too low and static transferrin level (hypotransferrinemia) with high transferrin saturation (usually > 55 %) and low ferritin value. Multiple tests are obligatory due to physiologically induced fluctuations.

5. Iron overload - Wikipedia

   en.wikipedia.org/wiki/Iron_overload

   Serum ferritin testing is a low-cost, readily available, and minimally invasive method for assessing body iron stores. However ferritin levels may be elevated due to a variety of other causes including obesity, infection, inflammation (as an acute phase protein), chronic alcohol intake, liver disease, kidney disease, and cancer.

6. Hemosiderosis - Wikipedia

   en.wikipedia.org/wiki/Hemosiderosis

   Serum ferritin is a low cost, readily available, and minimally invasive method for assessing body iron stores. However, the major problem with using it as an indicator of hemosiderosis is that it can be elevated in a range of other medical conditions unrelated to iron levels including infection, inflammation, fever, liver disease, renal disease ...

7. Transferrin - Wikipedia

   en.wikipedia.org/wiki/Transferrin

   This explains why ferritin levels remain relative low in Hereditary hemochromatosis, while transferrin saturation is high. [23] [24] Transferrin and its receptor have been shown to diminish tumour cells when the receptor is used to attract antibodies. [16]