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Pyridinium perbromide (also called pyridinium bromide perbromide, pyridine hydrobromide perbromide, or pyridinium tribromide) is an organic chemical composed of a pyridinium cation and a tribromide anion. It can also be considered as a complex containing pyridinium bromide—the salt of pyridine and hydrogen bromide—with an added bromine (Br ...
Pyridinium refers to the cation [C 5 H 5 NH] +. It is the conjugate acid of pyridine. Many related cations are known involving substituted pyridines, e.g. picolines ...
Tribromide is the anion with the chemical formula Br 3 −, or salts containing it: . Tetrabutylammonium tribromide; Tetrabromophosphonium tribromide; Pyridinium perbromide; Sodium and potassium tribromides can be prepared by reacting NaBr or KBr with aqueous bromine.
The pyridinium cation is isoelectronic with benzene. Pyridinium p - toluenesulfonate (PPTS) is an illustrative pyridinium salt; it is produced by treating pyridine with p -toluenesulfonic acid . In addition to protonation , pyridine undergoes N-centred alkylation , acylation , and N -oxidation .
The Cornforth reagent (pyridinium dichromate or PDC) is a pyridinium salt of dichromate with the chemical formula [C 5 H 5 NH] 2 [Cr 2 O 7].This compound is named after the Australian-British chemist Sir John Warcup Cornforth (b. 1917) who introduced it in 1962.
Pyridinium p-toluenesulfonate (PPTS) is a colourless solid salt of pyridine and p-toluenesulfonic acid. Uses. In organic synthesis, PPTS is used as a weakly acidic ...
The main use for phosphorus tribromide is for conversion of primary or secondary alcohols to alkyl bromides, [9] as described above.PBr 3 usually gives higher yields than hydrobromic acid, and it avoids problems of carbocation rearrangement- for example even neopentyl bromide can be made from the alcohol in 60% yield.
MPP + (1-methyl-4-phenylpyridinium) is a positively charged organic molecule with the chemical formula C 12 H 12 N +.It is a monoaminergic neurotoxin that acts by interfering with oxidative phosphorylation in mitochondria by inhibiting complex I, leading to the depletion of ATP and eventual cell death.