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[1] Hence, it is the biological basis for learning and the formation of new memories. [ 1 ] [ 2 ] Activity-dependent plasticity is a form of neuroplasticity that arises from intrinsic or endogenous activity, as opposed to forms of neuroplasticity that arise from extrinsic or exogenous factors, such as electrical brain stimulation - or drug ...
Hebbian theory is a neuropsychological theory claiming that an increase in synaptic efficacy arises from a presynaptic cell's repeated and persistent stimulation of a postsynaptic cell. It is an attempt to explain synaptic plasticity , the adaptation of brain neurons during the learning process.
Bienenstock–Cooper–Munro (BCM) theory, BCM synaptic modification, or the BCM rule, named after Elie Bienenstock, Leon Cooper, and Paul Munro, is a physical theory of learning in the visual cortex developed in 1981.
The book is a collection of stories of doctors and patients showing that the human brain is capable of undergoing change, including stories of recovering use of paralyzed body parts, deaf people learning to hear, and others getting relief from pain using exercises to retrain neural pathways.
Jeffrey Schwartz and Sharon Begley, The Mind and the Brain: Neuroplasticity and the power of mental force, New York: Regan Books, 2002. ISBN 0-06-039355-6 . Jeffrey Schwartz, You Are Not Your Brain: The 4-Step Solution for Changing Bad Habits, Ending Unhealthy Thinking, and Taking Control of Your Life , New York: Avery, 2011.
In 1973, M. M. Taylor [1] suggested that if synapses were strengthened for which a presynaptic spike occurred just before a postsynaptic spike more often than the reverse (Hebbian learning), while with the opposite timing or in the absence of a closely timed presynaptic spike, synapses were weakened (anti-Hebbian learning), the result would be an informationally efficient recoding of input ...
Two molecular mechanisms for synaptic plasticity involve the NMDA and AMPA glutamate receptors. Opening of NMDA channels (which relates to the level of cellular depolarization) leads to a rise in post-synaptic Ca 2+ concentration and this has been linked to long-term potentiation, LTP (as well as to protein kinase activation); strong depolarization of the post-synaptic cell completely ...
Neural plasticity is the neuronal basis for changes in how the mind works, including learning, the formation of memory, and changes in intelligence. One well-studied form of plasticity is Long-Term Potentiation (LTP). [6] It refers to a change in neural connectivity as a result of high activation on both sides of a synaptic cleft.