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The clinical examination of the skin is used to diagnose and distinguish between the various forms of pityriasis lichenoides. For pityriasis lichenoides, a skin biopsy followed by a histopathologic examination is a confirmatory procedure. Histologically, interface dermatitis with a noticeable lymphocytic infiltrate is the hallmark of pityriasis ...
There is no standard treatment for pityriasis lichenoides chronica. Treatments may include ultraviolet phototherapy, sun exposure, oral antibiotics, and corticosteroid creams and ointments to treat rash and itching. [3] [5] One study identified the enzyme bromelain as an effective therapeutic option for pityriasis lichenoides chronica. [6]
Pityriasis lichenoides et varioliformis acuta is a disease of the immune system. It is the more severe version of pityriasis lichenoides chronica. The disease is characterized by rashes and small lesions on the skin. The disease is more common in males and usually occurs in young adulthood, although it has been seen in every age group and every ...
Pityriasis lichenoides chronica (chronic guttate parapsoriasis, chronic pityriasis lichenoides, dermatitis psoriasiformis nodularis, parapsoriasis chronica, parapsoriasis lichenoides chronica) Pityriasis lichenoides et varioliformis acuta (acute guttate parapsoriasis, acute parapsoriasis, acute pityriasis lichenoides, Mucha–Habermann disease ...
Pityriasis lichenoides et varioliformis acuta, a disease of the immune system; Pityriasis rosea, a type of skin rash Pityriasis circinata, Pityriasis rubra pilaris, reddish-orange patches (Latin: rubra) on the skin; Pityriasis versicolor, a skin eruption on the trunk and proximal extremities, usually caused by a fungus
A lichenoid eruption is a skin disease characterized by damage and infiltration between the epidermis and dermis. [1]Examples include lichen planus, lichen sclerosus and lichen nitidus.
Certain conditions, like lichen striatus (LS) and pityriasis lichenoides chronica (PLC), typically cause postinflammatory hypopigmentation as opposed to hyperpigmentation. Postinflammatory hypopigmentation can also result after cutaneous injuries caused by burns, irritants, and dermatological operations (such as chemical peels , dermabrasion ...
Interface dermatitis with lichenoid inflammation, not otherwise specified, can be caused by lichen planus-like keratosis, lichenoid actinic keratosis, lichenoid lupus erythematosus, lichenoid GVHD (chronic GVHD), pigmented purpuric dermatosis, pityriasis rosea, and pityriasis lichenoides chronica. [2]