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  2. Magnesium salicylate - Wikipedia

    en.wikipedia.org/wiki/Magnesium_salicylate

    [citation needed] It is also used to treat joint pain like arthritis, [2] [3] general back pain, [4] and headaches. [citation needed] It is found in a variety of over-the-counter (OTC) medications, most notably the brand Doan's Pills, as an anti-inflammatory, primarily for back-pain relief. Magnesium salicylate can be an effective OTC ...

  3. Ibuprofen - Wikipedia

    en.wikipedia.org/wiki/Ibuprofen

    Ibuprofen is a weaker anti-inflammatory agent than other NSAIDs. [10] Ibuprofen was discovered in 1961 by Stewart Adams and John Nicholson [12] while working at Boots UK Limited and initially marketed as Brufen. [13] It is available under a number of brand names including Advil, Motrin, and Nurofen.

  4. Ibuprofen/paracetamol - Wikipedia

    en.wikipedia.org/wiki/Ibuprofen/paracetamol

    Ibuprofen/paracetamol, sold under the brand name Combogesic among others, is a fixed-dose combination of two medications, ibuprofen, a non-steroidal anti-inflammatory drug (NSAID); and paracetamol (acetaminophen), an analgesic and antipyretic. [6] It is available as a generic medication. [9] [10] [11]

  5. Profen (drug class) - Wikipedia

    en.wikipedia.org/wiki/Profen_(drug_class)

    The profens are a class of nonsteroidal anti-inflammatory drugs. [1] Profens are also known as 2-arylpropionic acids to reflect their chemical structure. [2] The most common example of a profen is ibuprofen, which has been sold under the brand name Profen among others. Other drugs in the class include: Alminoprofen; Benoxaprofen; Carprofen ...

  6. Damage to gut lining from ulcers, ibuprofen may increase ...

    www.aol.com/damage-gut-lining-ulcers-ibuprofen...

    A new study has suggested that damage to the upper gastrointestinal tract from conditions such as reflux, peptic ulcers, and prolonged use of NSAIDS may increase Parkinson’s risk by 76%.

  7. Anti-inflammatory - Wikipedia

    en.wikipedia.org/wiki/Anti-inflammatory

    Nonsteroidal anti-inflammatory drugs (NSAIDs) alleviate pain by counteracting the cyclooxygenase (COX) enzyme. [1] On its own, COX enzyme synthesizes prostaglandins, creating inflammation. In whole, the NSAIDs prevent the prostaglandins from ever being synthesized, reducing or eliminating the inflammation and resulting pain. [citation needed]

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