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The vestibular nerve is one of the two branches of the vestibulocochlear nerve (the cochlear nerve being the other). In humans the vestibular nerve transmits sensory information from vestibular hair cells located in the two otolith organs (the utricle and the saccule ) and the three semicircular canals via the vestibular ganglion of Scarpa .
Chances of recovery appear to decrease when treatment is delayed. Delay of treatment may result in permanent facial nerve paralysis. However, some studies demonstrate that even when steroids are started promptly, only 22% of all patients achieve full recovery of facial paralysis. [9] Treatment apparently has no effect on the recovery of hearing ...
There are three main patterns of facial nerve compression. The type of injury also gives an idea about the prognosis. Neuropraxia: no wallerian degeneration and complete and rapid recovery of function. Axonotmesis: wallerian degeneration and necrosis of the distal segment (death of the part of the nerve after the compression). Recovery is not ...
Vestibular rehabilitation (VR), also known as vestibular rehabilitation therapy (VRT), is a specialized form of physical therapy used to treat vestibular disorders or symptoms, characterized by dizziness, vertigo, imbalance, posture, and vision. These primary symptoms can result in secondary symptoms such as nausea, fatigue, and difficulty ...
Problems with balance can occur when there is a disruption in any of the vestibular, visual, or proprioceptive systems. Abnormalities in balance function may indicate a wide range of pathologies from causes like inner ear disorders, low blood pressure, brain tumors, and brain injury including stroke.
Vestibular neuritis is inflammation of the vestibular nerve (the nerve in the ear that sends messages related to motion and position to the brain). [2] [3] [4] Both conditions involve inflammation of the inner ear. [5] Labyrinths that house the vestibular system sense changes in the head's position or the head's motion. [6]
Vestibulopathies are disorders of the inner ear.They may include bilateral vestibulopathy, [1] central vestibulopathy, [2] post traumatic vestibulopathy, [3] peripheral vestibulopathy, [4] recurrent vestibulopathy, [5] visual vestibulopathy, [6] and neurotoxic vestibulopathy, [7] among others.
Other suggested causes of vestibular migraines include the following: unilateral neuronal instability of the vestibular nerve, idiopathic asymmetric activation of the vestibular nuclei in the brainstem, and vasospasm of the blood vessels supplying the labyrinth or central vestibular pathways resulting in ischemia to these structures. [21]