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These are common presenting symptoms of chronic and cardiogenic pulmonary edema due to left ventricular failure. The development of pulmonary edema may be associated with symptoms and signs of "fluid overload" in the lungs; this is a non-specific term to describe the manifestations of right ventricular failure on the rest of the body.
Orthopnea or orthopnoea [1] is shortness of breath (dyspnea) that occurs when lying flat, [2] causing the person to have to sleep propped up in bed or sitting in a chair. It is commonly seen as a late manifestation of heart failure, resulting from fluid redistribution into the central circulation, causing an increase in pulmonary capillary pressure and causing difficulty in breathing.
The hepatopulmonary syndrome results from the formation of microscopic intrapulmonary arteriovenous dilatations in patients with both chronic, and far less commonly acute liver failure. The mechanism is unknown but is thought to be due to increased liver production or decreased liver clearance of vasodilators, possibly involving nitric oxide. [1]
A parapneumonic effusion (circled), due to a left lower lobe pneumonia. A parapneumonic effusion is a type of pleural effusion (accumulation of fluid in the pleural cavity ) that arises as a result of a pneumonia , lung abscess , or bronchiectasis . [ 1 ]
A pulmonary consolidation is a region of normally compressible lung tissue that has filled with liquid instead of air. [1] The condition is marked by induration [2] (swelling or hardening of normally soft tissue) of a normally aerated lung. It is considered a radiologic sign.
People with respiratory failure often exhibit other signs or symptoms that are associated with the underlying cause of their respiratory failure. For instance, if respiratory failure is caused by cardiogenic shock (decreased perfusion due to heart dysfunction, symptoms of heart dysfunction (e.g., pitting edema) are also expected. Clubbing
A common response cascade to a variety of irritant gases includes inflammation, edema and epithelial sloughing, which if left untreated can result in scar formation and pulmonary and airway remodeling. Currently, mechanical ventilation remains the therapeutic mainstay for pulmonary dysfunction following acute inhalation injury.
In 1994, a new definition was recommended by the American-European Consensus Conference Committee [6] [10] which recognized the variability in severity of pulmonary injury. [51] The definition required the following criteria to be met: acute onset, persistent dyspnea; bilateral infiltrates on chest radiograph consistent with pulmonary edema