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Paragonimus westermani (Japanese lung fluke or oriental lung fluke) is the most common species of lung fluke that infects humans, causing paragonimiasis. [2] Human infections are most common in eastern Asia and in South America. Paragonimiasis may present as a sub-acute to chronic inflammatory disease of the lung. It was discovered by Dutch ...
Paragonimiasis is a food-borne parasitic disease caused by several species of lung flukes belonging to genus Paragonimus. [4] Infection is acquired by eating crustaceans such as crabs and crayfishes which host the infective forms called metacercariae, or by eating raw or undercooked meat of mammals harboring the metacercariae from crustaceans.
In the intestine, the parasite will move into the abdomen and commonly into the lungs. In the lung, the parasites encyst and cross fertilize each other. The cyst eventually ruptures in the lungs and the eggs may be coughed up or swallowed and excreted in the feces. An egg landing in fresh water hatches and releases a ciliated miracidium.
Clonorchis and Opisthorchis are carcinogenic parasites that are strongly associated with the development of cancer of the bile ducts. [8] [9] Lung flukes: there are ten species of lung flukes that infect humans, causing paragonimiasis. [10] Of these, the most common cause of human paragonimiasis is Paragonimus westermani, the oriental lung ...
urinary blood fluke Schistosoma haematobium: kidney, bladder, ureters, lungs, skin urine Africa, Middle East skin exposure to water contaminated with infected Bulinus sp. snails Schistosomiasis by Schistosoma japonicum: Schistosoma japonicum: intestine, liver, spleen, lungs, skin stool China, East Asia, Philippines
The lung fluke encysts within the tissue of crustaceans until the crustacean is consumed by humans or other animals. Once the fluke has been ingested, enzymes within the digestive tract of the consumer break down the parasitic cysts. The immature parasite continues to mature within the lungs of its new host, feeding on its intestine, and lay eggs.
The parasites subsequently invade deeper into the brain tissue, causing specific localizing neurological symptoms depending on where in the brain parenchyma they migrate. Neurologic findings and symptoms wax and wane as initial damage is done by the physical in-migration of the worms and secondary damage is done by the inflammatory response to ...
Fasciola hepatica, a species of liver flukes, has a higher incidence rate in children and females, with more cases of lung fluke and intestinal trematodiases in children. [5] Cases of liver and lung fluke trematodiasis are frequent due to the length of time the trematode can live in host organisms, and increased chances of reinfection. [9]
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