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Remyelination is the process of propagating oligodendrocyte precursor cells to form oligodendrocytes to create new myelin sheaths on demyelinated axons in the Central nervous system (CNS). This is a process naturally regulated in the body and tends to be very efficient in a healthy CNS. [ 1 ]
A remarkable finding in MS is that some Follicle-like aggregates appear in the meninges (composed by B-cells mostly infected with EBV [4]). These aggregates grow during the disease process and is mostly found in secondary progressive patients. Inflammation in the meninges has been found to be associated to gray mater (cortical) demyelination.
Myelinogenesis thus encompasses the process of transition between phases 3 and 4. [6] Upon initiation of myelinogenesis, each pioneer process forms lamellar extensions which extend and elaborate circumferentially around the target axon. This forms the first turn of the myelin sheath. [7]
A repair process, called remyelination, takes place in early phases of the disease, but the oligodendrocytes are unable to completely rebuild the cell's myelin sheath. Repeated attacks lead to successively less effective remyelinations, until a scar-like plaque is built up around the damaged axons.
10682 13595 Ensembl ENSG00000147155 ENSMUSG00000031168 UniProt Q15125 P70245 RefSeq (mRNA) NM_006579 NM_007898 RefSeq (protein) NP_006570 NP_031924 Location (UCSC) Chr X: 48.52 – 48.53 Mb Chr X: 8.05 – 8.06 Mb PubMed search Wikidata View/Edit Human View/Edit Mouse Emopamil binding protein is a protein that in humans is encoded by the EBP gene, located on the X chromosome. The protein is ...
A federal appeals court ruled that the Justice Department can release a report on Donald Trump’s efforts to overturn his 2020 election loss, but kept in place a judge's order requiring a three ...
As a noun, this word refers to a book that contains maps and charts. OK, that's it for hints—I don't want to totally give it away before revealing the answer! Related: ...
A repair process, called remyelination, takes place in the early phases of the disease, but the oligodendrocytes are unable to completely rebuild the cell's myelin sheath. [90] Repeated attacks lead to successively less effective remyelinations, until a scar-like plaque is built up around the damaged axons. [ 90 ]