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[9] [34] Dietary recommendations for cancer prevention typically include an emphasis on vegetables, fruit, whole grains, and fish, and avoidance of processed meat, red meat, animal fats, and refined carbohydrates. [35] The evidence to support these dietary changes is not definitive. [36]
Dietary recommendations for cancer prevention typically include weight management and eating a healthy diet, consisting mainly of "vegetables, fruit, whole grains and fish, and a reduced intake of red meat, animal fat, and refined sugar." [1] A healthy dietary pattern may lower cancer risk by 10–20%. [12]
Foods and drinks that promote weight gain: Limit consumption of energy-dense foods; Avoid sugary drinks. As calorie consumption is one of the harder tasks when it comes to monitoring weight-gain, it is a very important component in reducing the risk of cancer. Foods that have been processed heavily tend to contain more sugar and fat.
New research adds to the growing body of evidence suggesting that environmental factors, including the consumption of red meat and sugar, may be driving cases of colorectal cancer among young people.
In cancer cells, major changes in gene expression increase glucose uptake to support their rapid growth. Unlike normal cells, which produce lactate only when oxygen is low, cancer cells convert much of the glucose to lactate even in the presence of adequate oxygen. This is known as the “Warburg Effect.”
Scientist Otto Warburg, whose research activities led to the formulation of the Warburg hypothesis for explaining the root cause of cancer.. The Warburg hypothesis (/ ˈ v ɑːr b ʊər ɡ /), sometimes known as the Warburg theory of cancer, postulates that the driver of carcinogenesis (cancer formation) is insufficient cellular respiration caused by insult (damage) to mitochondria. [1]
Instead, choose more whole foods and make a point to get in good sources of omega-3s each week by eating fatty fish like salmon, canned tuna or mackerel, as well as walnuts, flaxseeds and chia seeds.
Some FODMAPs, such as fructose, are readily absorbed in the small intestine of humans via GLUT receptors. [19] Absorption thus depends on the appropriate expression and delivery of these receptors in the intestinal enterocyte to both the apical surface, contacting the lumen of the intestine (e.g., GLUT5), and to the basal membrane, contacting the blood (e.g., GLUT2). [19]