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Viral cardiomyopathy occurs when viral infections cause myocarditis with a resulting thickening of the myocardium and dilation of the ventricles. These viruses include Coxsackie B and adenovirus, echoviruses, influenza H1N1, Epstein–Barr virus, rubella (German measles virus), varicella (chickenpox virus), mumps, measles, parvoviruses, yellow fever, dengue fever, polio, rabies, and the ...
Dilated cardiomyopathy is when the muscle walls of your heart become stretched and can’t pump blood properly. It can be caused by inherited genes, chronic conditions, and unhealthy lifestyle habits.
Viral myocarditis being an outcome of viral infection depends heavily on genetic host factors and the pathogenicity unique to the virus. [56] If one tests positive for an acute viral infection, clinical developments have discovered that 1-5% of said population may show some form of myocarditis.
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Hypertrophic cardiomyopathy. Certain genes or other causes result in thickening of the heart chamber walls. Restrictive cardiomyopathy. This rare type causes scar tissue to replace normal heart ...
He added that symptoms of cardiomyopathy can also resemble other medical issues, which can lead to misdiagnosis, especially in younger patients who don’t fit the typical profile for heart failure.
Viral-induced dilated cardiomyopathy can be characterized using different methods. A 2011 study showed in coxsackievirus infected heart proteome , increased levels of fibrotic extracellular matrix proteins and reduced amounts of energy-producing enzymes can be observed suggesting they could be characteristic in enteroviral cardiomyopathy.
In 2015 cardiomyopathy and myocarditis affected 2.5 million people. [6] Hypertrophic cardiomyopathy affects about 1 in 500 people while dilated cardiomyopathy affects 1 in 2,500. [3] [10] They resulted in 354,000 deaths up from 294,000 in 1990. [7] [11] Arrhythmogenic right ventricular dysplasia is more common in young people. [2]
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