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ST segment depression and T-wave changes may be seen in patients with unstable angina; Depressed but upsloping ST segment generally rules out ischemia as a cause. Also, it can be a normal variant or artifacts, such as: Pseudo-ST-depression, which is a wandering baseline due to poor skin contact of the electrode [3]
ST elevation ≥1 mm in a lead with a positive QRS complex (i.e.: concordance) - 5 points; concordant ST depression ≥1 mm in lead V1, V2, or V3 - 3 points; ST elevation ≥5 mm in a lead with a negative (discordant) QRS complex - 2 points; ≥3 points = 90% specificity of STEMI (sensitivity of 36%) [2]
Schematic representation of normal ECG. In electrocardiography, the ST segment connects the QRS complex and the T wave and has a duration of 0.005 to 0.150 sec (5 to 150 ms). It starts at the J point (junction between the QRS complex and ST segment) and ends at the beginning of the T wave.
These two leads, ST depression will be seen because they are the opposing leads of the cardiac axis. PR segment depression is highly suggestive of pericarditis. R wave in most cases will be unaltered. In two weeks after pericarditis, there will be upward concave ST elevation, positive T wave, and PR depression.
those with ST segment depression or T wave inversion (suspicious for ischemia), and; those with a so-called non-diagnostic or normal ECG. A normal ECG does not rule out acute myocardial infarction. Mistakes in interpretation are relatively common, and the failure to identify high risk features has a negative effect on the quality of patient ...
Diagnosis is based on an ECG showing ST-segment depression at the J-point of 1 to 3 mm in leads V1 to V6, with tall and symmetrical T waves. [1] The ST-segment is upsloping and there is also often ST-segment elevation of 0.5 to 2 mm in lead aVR. [1] [2] The QRS complex is either normal or slightly wide. [1] Treatment is as per an ST elevation ...
The ECG should be done as early as practicable, including in the ambulance if possible. [18] ECG changes indicating acute heart damage include: ST elevation, new left bundle branch block and ST depression amongst others. The absence of ECG changes does not immediately distinguish between unstable angina and NSTEMI. [6]
On the surface ECG, premature junctional contractions will appear as a normally shaped ventricular complex or QRS complex, not preceded by any atrial complex or P wave or preceded by an abnormal P wave with a shorter PR interval. Rarely, the abnormal P wave can follow the QRS. [2]