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Despite widely variable intervals between meals or the occasional consumption of meals with a substantial carbohydrate load, human blood glucose levels tend to remain within the normal range. However, shortly after eating, the blood glucose level may rise, in non-diabetics, temporarily up to 7.8 mmol/L (140 mg/dL) or slightly more.
"If you're healthy and consume a normal diet, you can expect your blood sugar level to be between 100 and 130 mg/dL after eating." ... "Alcohol raises blood glucose levels, so it's [also] best to ...
Hyperglycemia or hyperglycaemia is a condition where unusually high amount of glucose is present in blood. It is defined as blood glucose level exceeding 6.9 mmol/L (125 mg/dL) after fasting for 8 hours and 10 mmol/L (180 mg/dL) 2 hours after eating. [1] [2]
The consequent fall in blood glucose is indicated as the reason for the "sugar crash". [4] Another cause might be hysteresis effect of insulin action, i.e., the effect of insulin is still prominent even if both plasma glucose and insulin levels were already low, causing a plasma glucose level eventually much lower than the baseline level. [5]
Soda and juices wreak havoc on blood-sugar levels. Add some interest to seltzer by squeezing in fresh citrus, opting for flavored (but not sweetened) versions, or infusing with a sprig of fresh herbs.
After people eat, it’s completely normal for their blood sugar to rise, Natter said. However, it can be a problem if the number rises too high. Surampudi said, “If someone is not diabetic ...
The glycemic response (or glycaemic response) to a food or meal is the effect that food or meal has on blood sugar (glucose) levels after consumption. [1] It is normal for blood glucose and insulin levels to rise after eating and then return again to fasting levels over a short period of time.
GLP-1 and DPP-4 inhibitors. Incretins are a group of metabolic hormones that stimulate a decrease in blood glucose levels. Incretins are released after eating and augment the secretion of insulin released from pancreatic beta cells of the islets of Langerhans by a blood-glucose–dependent mechanism.
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