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Paragonimus westermani (Japanese lung fluke or oriental lung fluke) is the most common species of lung fluke that infects humans, causing paragonimiasis. [2] Human infections are most common in eastern Asia and in South America. Paragonimiasis may present as a sub-acute to chronic inflammatory disease of the lung. It was discovered by Dutch ...
Paragonimiasis is a food-borne parasitic disease caused by several species of lung flukes belonging to genus Paragonimus. [4] Infection is acquired by eating crustaceans such as crabs and crayfishes which host the infective forms called metacercariae, or by eating raw or undercooked meat of mammals harboring the metacercariae from crustaceans.
In the intestine, the parasite will move into the abdomen and commonly into the lungs. In the lung, the parasites encyst and cross fertilize each other. The cyst eventually ruptures in the lungs and the eggs may be coughed up or swallowed and excreted in the feces. An egg landing in fresh water hatches and releases a ciliated miracidium.
The lung fluke encysts within the tissue of crustaceans until the crustacean is consumed by humans or other animals. Once the fluke has been ingested, enzymes within the digestive tract of the consumer break down the parasitic cysts. The immature parasite continues to mature within the lungs of its new host, feeding on its intestine, and lay eggs.
Main article: Human parasite Endoparasites Protozoan organisms Common name of organism or disease Latin name (sorted) Body parts affected Diagnostic specimen Prevalence Source/Transmission (Reservoir/Vector) Granulomatous amoebic encephalitis and Acanthamoeba keratitis (eye infection) Acanthamoeba spp. eye, brain, skin culture worldwide contact lenses cleaned with contaminated tap water ...
Sparganosis is a parasitic infection caused by the plerocercoid larvae of the genus Spirometra including S. mansoni, S. ranarum, S. mansonoides and S. erinacei. [1] [2] It was first described by Patrick Manson in 1882, [3] and the first human case was reported by Charles Wardell Stiles from Florida in 1908. [4]
The general life cycle of a lungworm begins with an ingestion of infective larvae. The infective larvae then penetrate the intestinal wall where larvae migrate into the lungs through the bloodstream. The infected larvae reside in the lungs until the development into an adult lungworm. The eggs of the adult hatch thus producing L1 larvae.
Fasciola hepatica, a species of liver flukes, has a higher incidence rate in children and females, with more cases of lung fluke and intestinal trematodiases in children. [5] Cases of liver and lung fluke trematodiasis are frequent due to the length of time the trematode can live in host organisms, and increased chances of reinfection. [9]