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A death rattle is noisy breathing that often occurs in someone near death. [1] Accumulation of fluids such as saliva and bronchial secretions in the throat and upper airways is the cause. [ 2 ] Those who are dying may lose their ability to swallow and may have increased production of bronchial secretions, resulting in such an accumulation. [ 3 ]
Symptoms tend to be brief, but can range from mild to severe and include: fever, vomiting, increased respirations or difficulty breathing, cough, wheeze, and rash. Symptoms typically follow an exposure to allergens or certain drugs, and last approximately two weeks. [3] Eosinophilia is the main feature of diagnostic criteria for Loffler's syndrome.
That is, in whispered pectoriloquy, the repeated words are whispered at low volume, and in bronchophony, they are spoken at normal volume. The clinical observation being determined is whether or not an increase in volume is heard at the clinician's stethoscope over the lung field being auscultated which would indicate lung consolidation.
In Susac's syndrome, an autoimmune response damages the blood vessels of the brain, retina and cochlea, leading to a loss of neurological functions. Patients with the syndrome had hearing and vision loss and were found to have higher concentrations of lymphocytes and proteins in their cerebrospinal fluid.
Lymphoproliferative disorders are a set of disorders characterized by the abnormal proliferation of lymphocytes into a monoclonal lymphocytosis. The two major types of lymphocytes are B cells and T cells, which are derived from pluripotent hematopoietic stem cells in the bone marrow.
The genetic cause of Bare lymphocyte syndrome type II is due to mutations in any of the following genes: [5]. CIITA is responsible for giving instructions to create a protein that controls transcription of genes (MHC class II), and is located at 16p13.13 (cytogenetic location), [6]
The cause of TRALI is currently not fully understood. 80–85% of cases are thought to be immune mediated. [5] [6] Antibodies directed toward human leukocyte antigens (HLA) or human neutrophil antigens (HNA) have been implicated, with transfused antibodies shown to bind antigens expressed on pulmonary endothelial cells to initiate acute inflammation in the lungs.
Preservation of lung volumes in the presence of increased interstitial markings is a radiographic hallmark of LAM that helps distinguish it from most other interstitial lung diseases, in which alveolar septal and interstitial expansion tend to increase the lung's elastic recoil properties and decreased lung volumes.