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Side effects from vitamin overdoses may occur, especially in patients with kidney failure. In very large doses, vitamin C in kidney-failure patients can cause severe side effects such as oxalate deposits in bone and soft tissues and may interfere with the absorption and metabolism of vitamin B 12. The product contains 1 g of vitamin C per ...
Citric acid sold in a dry powdered form is commonly sold in markets and groceries as "sour salt", due to its physical resemblance to table salt. It has use in culinary applications, as an alternative to vinegar or lemon juice, where a pure acid is needed. Citric acid can be used in food coloring to balance the pH level of a normally basic dye.
Disodium citrate, also known as disodium hydrogen citrate, (Neo-Alkacitron) and sesquihydrate, is an acid salt of citric acid with the chemical formula Na 2 C 6 H 6 O 7. [2] It is used as an antioxidant in food and to improve the effects of other antioxidants.
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If there is no oxygen available for the parts of the glucose metabolism that require oxygen (citric acid cycle and oxidative phosphorylation), excess pyruvate will be converted in excess lactate. In "type B" lactic acidosis the lactate accumulates because there is a mismatch between glycolysis activity and the remainder of glucose metabolism.
Citric acid/potassium-sodium citrate is a drug used in the treatment of metabolic acidosis (a disorder in which the blood is too acidic). It is made up of citrate (the weak base of citric acid), a sodium cation and potassium cation. It can also be used for the treatment of kidney stones [1] by treating hypocitraturia. [2]
Drugs used in diabetes treat types of diabetes mellitus by decreasing glucose levels in the blood.With the exception of insulin, most GLP-1 receptor agonists (liraglutide, exenatide, and others), and pramlintide, all diabetes medications are administered orally and are thus called oral hypoglycemic agents or oral antihyperglycemic agents.
The Randle cycle, also known as the glucose fatty-acid cycle, is a metabolic process involving the cross inhibition of glucose and fatty acids for substrates. [1] It is theorized to play a role in explaining type 2 diabetes and insulin resistance. [2] [3] It was named for Philip Randle, who described it in 1963. [4]
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