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There was a statistically significant difference between the number of patients in the treatment group]and control group at the 30% reduction in pain level using the Visual Analogue Scale (VAS). In addition the study showed that, on average, the treatment group (device on) had 6.1 fewer days of headache days per month which was more than double ...
Occipital neuralgia is caused by damage to the occipital nerves, which can arise from trauma (usually concussive or cervical), physical stress on the nerve, repetitive neck contraction, flexion or extension, and/or as a result of medical complications (such as osteochondroma, a benign bone tumour).
The greater occipital nerve is a nerve of the head. It is a spinal nerve, specifically the medial branch of the dorsal primary ramus of cervical spinal nerve 2.It arises from between the first and second cervical vertebrae, ascends, and then passes through the semispinalis muscle.
The lesser occipital nerve (or small occipital nerve [1]) is a cutaneous spinal nerve of the cervical plexus. [2] It arises from second cervical (spinal) nerve (C2) (along with the greater occipital nerve).
Undertreatment of pain is the absence of pain management therapy for a person in pain when treatment is indicated. Consensus in evidence-based medicine and the recommendations of medical specialty organizations establish guidelines to determine the treatment for pain which health care providers ought to offer. [91]
The occipitalis muscle (occipital belly) is a muscle which covers parts of the skull. Some sources consider the occipital muscle to be a distinct muscle. However, Terminologia Anatomica currently classifies it as part of the occipitofrontalis muscle along with the frontalis muscle. The occipitalis muscle is thin and quadrilateral in form.
Flexion is produced mainly by the action of the longi capitis and recti capitis anteriores; extension by the recti capitis posteriores major and minor, the obliquus capitis superior, the semispinalis capitis, splenius capitis, sternocleidomastoideus, and upper fibers of the trapezius.
The intensity of signal is increased in five to ten minute intervals until the patient feels a relief of pain or until the scrambler signal intensity begins to cause pain. This intensity of electrical stimulation is then continued. [6] Each treatment session lasts for about 30-40 minutes, and may be repeated on other days during the treatment ...
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