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A normal resting heart rate is 60 to 100 beats per minute. A resting heart rate of more than 100 beats per minute is defined as a tachycardia. During an episode of SVT, the heart beats about 150 to 220 times per minute. [9] Specific treatment depends on the type of SVT [5] and can include medications, medical procedures, or surgery. [5]
Atrial flutter is characterized by a sudden-onset (usually) regular abnormal heart rhythm on an electrocardiogram (ECG) in which the heart rate is fast. Symptoms may include a feeling of the heart beating too fast, too hard, or skipping beats, chest discomfort, difficulty breathing, a feeling as if one's stomach has dropped, a feeling of being ...
Both doctors say that signs and symptoms of valve damage can vary, but the main ones to be aware of are shortness of breath (particularly during exercise or any form of exertion) and chest pain or ...
Termination of PSVT following adenosine administration. Adenosine, an ultra-short-acting AV nodal blocking agent, is indicated if vagal maneuvers are not effective. [17] If unsuccessful or the PSVT recurs, calcium channel blockers, such as diltiazem or verapamil, are recommended. [4] Adenosine may be safely used during pregnancy. [18]
The main symptom of AVNRT is the sudden development of rapid regular palpitations. [1] These palpitations may be associated with a fluttering sensation in the neck, caused by near-simultaneous contraction of the atria and ventricles against a closed tricuspid valve leading to the pressure or atrial contraction being transmitted backwards into the venous system. [2]
[1] [5] People with TIC may have symptoms associated with heart failure (e.g. shortness of breath or ankle swelling) and/or symptoms related to the tachycardia or arrhythmia (e.g. palpitations). [ 1 ] [ 2 ] Though atrial fibrillation is the most common cause of TIC, several tachycardias and arrhythmias have been associated with the disease.
If these maneuvers fail, using intravenous adenosine [4] causes complete electrical blockade at the AV node and interrupts the reentrant electrical circuit. Long-term management includes beta blocker therapy and radiofrequency ablation of the accessory pathway.
Often, symptoms mimic those of congestive heart failure (esp. activity intolerance and dyspnea), but treatment of each is different. Beta blockers are used in both cases, but treatment with diuretics, a mainstay of CHF treatment, will exacerbate symptoms in hypertrophic obstructive cardiomyopathy by decreasing ventricular preload volume and ...
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