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Infants with IUGR are also at elevated risk for neurodevelopmental abnormalities, including motor delay and cognitive impairments. Low IQ in adulthood may occur in up to one third of infants born small for gestational age due to IUGR. Infants who fail to display adequate catch-up growth in the first few years of life may exhibit worse outcomes.
Stunted growth, also known as stunting or linear growth failure, is defined as impaired growth and development manifested by low height-for-age. [1] It is a manifestation of malnutrition and can be caused by endogenous factors (such as chronic food insecurity ) or exogenous factors (such as parasitic infection ).
Other symptoms include a heart murmur which may range from almost imperceptible to very loud, difficulty in feeding, failure to gain weight, retarded growth and physical development, labored breathing (dyspnea) on exertion, clubbing of the fingers and toes, and polycythemia. [2] The baby may turn blue with breastfeeding or crying. [2]
Several conditions and risk factors can lead to fetal distress or non-reassuring fetal status, [1] including: Low amniotic fluid (oligohydramnios) If there is too little amniotic fluid around the baby in the uterus, the baby can have trouble moving around in the uterus and its growth and temperature can be impacted.
Congenital iodine deficiency syndrome (CIDS), also called cretinism, [2] is a medical condition present at birth marked by impaired physical and mental development, due to insufficient thyroid hormone production (hypothyroidism) often caused by insufficient dietary iodine during pregnancy.
English: Stages in Prenatal development, with weeks and months numbered by gestation. Image made in Inkscape. References are found in equivalent Wikipedia articles: Fetus; Gestational age; Human development (biology) Pregnancy; Prenatal development; Viability (fetal)
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Intrauterine hypoxia can be attributed to maternal, placental, or fetal conditions. [12] Kingdom and Kaufmann classifies three categories for the origin of fetal hypoxia: 1) pre-placental (both mother and fetus are hypoxic), 2) utero-placental (mother is normal but placenta and fetus is hypoxic), 3) post-placental (only fetus is hypoxic).
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