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Platypnea or platypnoea is shortness of breath (dyspnea) that is relieved when lying down, and worsens when sitting or standing upright. It is the opposite of orthopnea. [1] The condition was first described in 1949 and named in 1969.
Many different conditions can lead to the feeling of dyspnea (shortness of breath). DiagnosisPro, an online medical expert system, listed 497 in October 2010. [1] The most common cardiovascular causes are acute myocardial infarction and congestive heart failure while common pulmonary causes include: chronic obstructive pulmonary disease, asthma, pneumothorax, and pneumonia.
Shortness of breath (SOB), known as dyspnea (in AmE) or dyspnoea (in BrE), is an uncomfortable feeling of not being able to breathe well enough. The American Thoracic Society defines it as "a subjective experience of breathing discomfort that consists of qualitatively distinct sensations that vary in intensity", and recommends evaluating dyspnea by assessing the intensity of its distinct ...
Shortness of breath, jaw pain, and fatigue are common symptoms that can signal an underlying medical condition. Dr. Natalie Azar tells us when to see a doctor.
An anginal equivalent is a symptom such as shortness of breath , diaphoresis (sweating), extreme fatigue, or pain at a site other than the chest, occurring in a patient at high cardiac risk. Anginal equivalents are considered to be symptoms of myocardial ischemia .
Pneumonia is a lung infection that can cause pain under the breast when you inhale along with other symptoms like cough, shortness of breath, and fever, says Dr. Ross.
The pain may stay in one place, or it may spread to the shoulder or back. [10] Sometimes, it becomes a fairly constant dull ache. [11] Depending on its cause, pleuritic chest pain may be accompanied by other symptoms: [12] Dry cough; Fever and chills; Rapid, shallow breathing; Shortness of breath; Fast heart rate
Causes may include heart failure, kidney failure, narcotic poisoning, intracranial pressure, and hypoperfusion of the brain (particularly of the respiratory center). The pathophysiology of Cheyne–Stokes breathing can be summarized as apnea leading to increased CO 2 which causes excessive compensatory hyperventilation, in turn causing decreased CO 2 which causes apnea, restarting the cycle.