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Homologous desensitization also occurs with cytokine and other types of receptors, such as those of the epidermal growth factor receptor type, but in these cases desensitization is mediated by other types of receptor kinases. [2] Homologous desensitization serves to limit or restrain a cell's responses to stimuli.
In medicine, desensitization is a method to reduce or eliminate an organism's negative reaction to a substance or stimulus. In pharmacology , drug desensitization refers to two related concepts. First, desensitization may be equivalent to drug tolerance and refers to subjects' reactions (positive or negative) to a drug reducing following its ...
Medications are usually not needed as hand, foot, and mouth disease is a viral disease that typically resolves on its own. Under research [15] [16] Sin Nombre virus: Hantavirus Pulmonary Syndrome (HPS) No Heartland virus: Heartland virus disease No Helicobacter pylori: Helicobacter pylori infection No Escherichia coliO157:H7, O111 and O104:H4
A list of examples of in vivo transdifferentiation through transfection: [1] mouse hepatocytes → B cells (Pdx1) [2] exocrine cells → B cells (Pdx1, Ngn3, and v-maf musculoaponeurotic fibrosarcoma oncogene family protein A) [3] nonsensory cells → inner hair cells (Atoh1 and MathI) [4]
Allergen immunotherapy, also known as desensitization or hypo-sensitization, is a medical treatment for environmental allergies (such as insect bites) and asthma. [ 1 ] [ 2 ] Immunotherapy involves exposing people to larger and larger amounts of allergens in an attempt to change the immune system's response.
For example, PDE compromised the antiepileptic activity of adenosine. In addition, using of a PDE inhibitor (pentoxifylline) in pentylenetetrazole-induced seizure indicated the antiepileptic effect by increasing the time latency to seizure incidence and decreasing the seizure duration in vivo.
Homologous desensitization occurs when a receptor decreases its response to an agonist at high concentration. [1] It is a process through which, after prolonged agonist exposure, the receptor is uncoupled from its signaling cascade and thus the cellular effect of receptor activation is attenuated.
Since the 1950s, several sorts of exposure therapy have been developed, including systematic desensitization, flooding, implosive therapy, prolonged exposure therapy, in vivo exposure therapy, and imaginal exposure therapy. [22] Exposure and response prevention (ERP) traces its roots back to the work of psychologist Vic Meyer in the 1960s.