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The coracoid process acts as an attachment and origin for a large number of muscles (attached muscles not labeled here). The coracoid process is a thick curved process attached by a broad base to the upper part of the neck of the scapula; [2] it runs at first upward and medially; then, becoming smaller, it changes its direction, and projects forward and laterally.
The coracohumeral ligament arises from the lateral border or the base of the coracoid process. [1] [2] It passes obliquely downwards and laterally to the front of the greater tubercle of the humerus. [1] [2] It forms two bands [3]: 908 - an anterior one and a posterior one - that insert into the lesser and greater tubercles of the humerus ...
A coracoid [a] is a paired bone which is part of the shoulder assembly in all vertebrates except therian mammals (marsupials and placentals). In therian mammals (including humans ), a coracoid process is present as part of the scapula , but this is not homologous with the coracoid bone of most other vertebrates.
The coracoacromial ligament is a strong triangular ligament between the coracoid process and the acromion. It protects the head of the humerus . Its acromial attachment may be repositioned to the clavicle during reconstructive surgery of the acromioclavicular joint (shoulder joint).
The coracoclavicular ligament connects the clavicle to the coracoid process of the scapula. [1] It is not part of the acromioclavicular joint articulation, but is usually described with it, since it keeps the clavicle in contact with the acromion. It consists of two fasciculi, the trapezoid ligament in front, and the conoid ligament behind. [2]
In human anatomy, the acromion (from Greek: akros, "highest", ōmos, "shoulder", pl.: acromia) is a bony process on the scapula (shoulder blade). Together with the coracoid process, it extends laterally over the shoulder joint. The acromion is a continuation of the scapular spine, and hooks over anteriorly.
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On imaging, it is defined by the coracoid process at its base, the supraspinatus tendon superiorly and the subscapularis tendon inferiorly. Changes of adhesive capsulitis can be seen at this interval as edema and fibrosis. Pathology at the interval is also associated with glenohumeral and biceps instability. [16]